Cellular response to DNA interstrand crosslinks: The Fanconi Anemia pathway
Interstrand crosslinks (ICLs) are a highly toxic form of DNA damage. ICLs can interfere with vital biological processes requiring separation of the two DNA strands, such as replication and transcription. If ICLs are left unrepaired, it can lead to mutations, chromosome breakage and mitotic catastrop...
Κύριοι συγγραφείς: | Lopez-Martinez, D, Liang, C, Cohn, M |
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Μορφή: | Journal article |
Έκδοση: |
Springer Verlag
2016
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Παρόμοια τεκμήρια
Παρόμοια τεκμήρια
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UHRF1 Is a sensor for DNA interstrand crosslinks and recruits FANCD2 to initiate the Fanconi Anemia pathway
ανά: Liang, C, κ.ά.
Έκδοση: (2015) -
UHRF1 Is a Sensor for DNA Interstrand Crosslinks and Recruits FANCD2 to Initiate the Fanconi Anemia Pathway
ανά: Chih-Chao Liang, κ.ά.
Έκδοση: (2015-03-01) -
UHRF1 is a sensor for DNA interstrand crosslinks.
ανά: Cohn, M, κ.ά.
Έκδοση: (2016) -
Elevated levels of STAT1 in Fanconi anemia group A lymphoblasts correlate with the cells’ sensitivity to DNA interstrand crosslinking drugs
ανά: Inés Prieto-Remón, κ.ά.
Έκδοση: (2013-05-01) -
Components of a Fanconi-like pathway control Pso2-independent DNA interstrand crosslink repair in yeast
ανά: Ward, T, κ.ά.
Έκδοση: (2012)