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Guanidinoacetate N-methyltrans...
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Guanidinoacetate N-methyltransferase-knockout mice completely lacking myocardial phosphocreatine show normal in vivo cardiac function, but increased susceptibility to ischemia/reperfusion injury
Bibliographic Details
Main Authors:
Lygate, C
,
ten Hove, M
,
Wallis, J
,
Fischer, A
,
Schneider, J
,
Sang, E
,
Dawson, D
,
Hulbert, K
,
Sebag-Montefiore, L
,
Cassidy, P
,
Watkins, H
,
Clarke, K
,
Isbrandt, D
,
Neubauer, S
Format:
Conference item
Published:
2004
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