Guanidinoacetate N-methyltransferase-knockout mice completely lacking myocardial phosphocreatine show normal in vivo cardiac function, but increased susceptibility to ischemia/reperfusion injury

Guanidinoacetate N-methyltransferase-knockout mice completely lacking myocardial phosphocreatine show normal in vivo cardiac function, but increased susceptibility to ischemia/reperfusion injury

Bibliographic Details
Main Authors:	Lygate, C, ten Hove, M, Wallis, J, Fischer, A, Schneider, J, Sang, E, Dawson, D, Hulbert, K, Sebag-Montefiore, L, Cassidy, P, Watkins, H, Clarke, K, Isbrandt, D, Neubauer, S
Format:	Conference item
Published:	2004

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