Impairment of cholinergic signaling reduces atrial beta-adrenergic responsiveness by enhancing eNOS expression

Impairment of cholinergic signaling reduces atrial beta-adrenergic responsiveness by enhancing eNOS expression

Manylion Llyfryddiaeth
Prif Awduron:	Zhang, Y, Danson, E, Sears, C, Edwards, A, Casadei, B, Paterson, D
Fformat:	Conference item
Cyhoeddwyd:	2004

Eitemau Tebyg

Disruption of inhibitory G-proteins mediates a reduction in atrial beta-adrenergic signaling by enhancing eNOS expression.
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Cyhoeddwyd: (2005)

nNOS V eNOS mediated regulation of beta adrenergic inotropic and lusitropic responses in left ventricular myocytes
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Cyhoeddwyd: (2004)

Are myocardial eNOS and nNOS involved in the beta-adrenergic and muscarinic regulation of inotropy? A systematic investigation.
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Myocardial NOS1 controls the lusitropic response to beta-adrenergic stimulation in vivo and in vitro
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Nitric oxide synthase 1 (NOS1) regulates basal and beta-adrenergic left-ventricular contraction and relaxation in vivo
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Cyhoeddwyd: (2003)

Nitric oxide synthase 1 (NOS1) regulates basal and beta-adrenergic left ventricular contraction and relaxation in vivo
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Cyhoeddwyd: (2003)

Neuronal nitric oxide synthase (NOS1) regulates beta-adrenergic contraction and calcium handling in isolated murine ventricular myocytes
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Cyhoeddwyd: (2003)

Reduced expression of eNOS in the fractured femoral neck
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Beta-adrenergic and M-cholinergic receptor interactions in the pathogenesis of bronchial obstructive pulmonary diseases
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Enhancement of beta adrenergic responsiveness in myocytes lacking nitric oxide synthase 1
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Cardiac nitric oxide synthase 1 regulates basal and beta-adrenergic contractility in murine ventricular myocytes.
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The negative inotropic effect of beta(3)-adrenergic receptor stimulation in nNOS(-/-) mice is restored by oxypurinol
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The Cholinergic and Adrenergic Autocrine Signaling Pathway Mediates Immunomodulation in Oyster Crassostrea gigas
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Sympathetic activation increases NO release from eNOS but neither eNOS nor nNOS play an essential role in exercise hyperemia in the human forearm.
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Cholinergic control of heart rate by nitric oxide is site specific.
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Reduced phospholamban phosphorylation is responsible for impaired myocardial relaxation in nNOS(-/-) mice
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Beta adrenergic blockade reduces utilitarian judgement.
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Reduced phospholamban phosphorylation is responsible for impaired myocardial relaxation in nNOS(-/-) mice
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mTOR contributes to endothelium-dependent vasorelaxation by promoting eNOS expression and preventing eNOS uncoupling
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Adrenergic/Cholinergic Immunomodulation in the Rat Model—In Vivo Veritas?
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The Interaction of Cholinergic Muscarinic and Beta-1 Adrenergic Receptors of the CA1 Region in Passive Avoidance Memory Formation in Rat
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Beta-Adrenergic Agonists
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Participation of cholinergic and adrenergic mechanisms in gastric reactions to exogenous serotonin infusion
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Pupil fluctuations track rapid changes in adrenergic and cholinergic activity in cortex
gan: Jacob Reimer, et al.
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Expression of eNOS in kidneys from hypertensive patients
gan: Xin Gu, et al.
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Cardiac cholinergic NO-cGMP signaling following acute myocardial infarction and nNOS gene transfer.
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Orientin Reduces Myocardial Infarction Size via eNOS/NO Signaling and Thus Mitigates Adverse Cardiac Remodeling
gan: Fangfang Li, et al.
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High-fat stimulation induces atrial neural remodeling by reducing NO production via the CRIF1/eNOS/P21 axi
gan: An Zhang, et al.
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The roles of beta-adrenergic receptors in tumorigenesis and the possible use of beta-adrenergic blockers for cancer treatment: possible genetic and cell-signaling mechanisms
gan: Luong KV, et al.
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Beta Adrenergic Signaling: A Targetable Regulator of Angiosarcoma and Hemangiosarcoma
gan: Erin B. Dickerson, et al.
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Myricetin ameliorated ischemia/reperfusion-induced brain endothelial permeability by improvement of eNOS uncoupling and activation eNOS/NO
gan: Song Zhang, et al.
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Beta adrenergic blockers poisoning
gan: Tadić Milorad, et al.
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eNOS in congenital to adult cardiovascular disease
gan: Kirsty M. Hooper
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Vascular nitric oxide: Beyond eNOS
gan: Yingzi Zhao, et al.
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PKC-dependent phosphorylation of eNOS at T495 regulates eNOS coupling and endothelial barrier function in response to G+ -toxins.
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Stoichiometric relationships between endothelial tetrahydrobiopterin, endothelial NO synthase (eNOS) activity, and eNOS coupling in vivo: insights from transgenic mice with endothelial-targeted GTP cyclohydrolase 1 and eNOS overexpression.
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Cyhoeddwyd: (2005)

Effects of inhibition of eNOS and nNOS on forearm blood flow during reflex sympathetic activation at rest and during exercise
gan: Shabeeh, H, et al.
Cyhoeddwyd: (2012)

Loss of cholinergic innervation differentially affects eNOS-mediated blood flow, drainage of Aβ and cerebral amyloid angiopathy in the cortex and hippocampus of adult mice
gan: Shereen Nizari, et al.
Cyhoeddwyd: (2021-01-01)

Neural control of coronary artery blood flow by non‐adrenergic and non‐cholinergic mechanisms
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Tetrahydrobiopterin- and glutathionylation-dependent regulation of eNOS coupling: Relationships revealed by eNOS glutathionylation mutants in cells with tet-regulated GTP cyclohydrolase I expression
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Cyhoeddwyd: (2012)