Control of established experimental allergic encephalomyelitis by inhibition of tumor necrosis factor (TNF) activity within the central nervous system using monoclonal antibodies and TNF receptor-immunoglobulin fusion proteins.

Ítems similars

• The effects of tumor necrosis factor (TNF) derivatives on TNF receptors.
  per: Tchorzewski, H, et al.
  Publicat: (1993)
• Gene therapy for chronic relapsing experimental allergic encephalomyelitis using cells expressing a novel soluble p75 dimeric TNF receptor.
  per: Croxford, J, et al.
  Publicat: (2000)
• Local gene therapy with CTLA4-immunoglobulin fusion protein in experimental allergic encephalomyelitis.
  per: Croxford, J, et al.
  Publicat: (1998)
• TNF receptor fusion proteins are effective inhibitors of TNF-mediated cytotoxicity on human KYM-1D4 rhabdomyosarcoma cells.
  per: Butler, D, et al.
  Publicat: (1994)
• Cloning of human tumor necrosis factor (TNF) receptor cDNA and expression of recombinant soluble TNF-binding protein.
  per: Gray, P, et al.
  Publicat: (1990)