The transcription factor Erg inhibits vascular inflammation by repressing NF-kappaB activation and proinflammatory gene expression in endothelial cells.

Eitemau Tebyg

• NF-kappaB-inducing kinase is dispensable for activation of NF-kappaB in inflammatory settings but essential for lymphotoxin beta receptor activation of NF-kappaB in primary human fibroblasts.
  gan: Smith, C, et al.
  Cyhoeddwyd: (2001)
• Activation of NF-kappaB by the intracellular expression of NF-kappaB-inducing kinase acts as a powerful vaccine adjuvant.
  gan: Andreakos, E, et al.
  Cyhoeddwyd: (2006)
• Is NF-kappaB a useful therapeutic target in rheumatoid arthritis?
  gan: Feldmann, M, et al.
  Cyhoeddwyd: (2002)
• Hydrogen peroxide prolongs nuclear localization of NF-kappaB in activated cells by suppressing negative regulatory mechanisms.
  gan: Enesa, K, et al.
  Cyhoeddwyd: (2008)
• NF-kappaB suppression by the deubiquitinating enzyme Cezanne: a novel negative feedback loop in pro-inflammatory signaling.
  gan: Enesa, K, et al.
  Cyhoeddwyd: (2008)