Subtype-specific actions of beta-amyloid peptides on recombinant human neuronal nicotinic acetylcholine receptors (alpha7, alpha4beta2, alpha3beta4) expressed in Xenopus laevis oocytes.

समान संसाधन

• The Abeta1-42M35C mutated amyloid peptide Abeta1-42 and the 25-35 fragment fail to mimic the subtype-specificity of actions on recombinant human nicotinic acetylcholine receptors (alpha7, alpha4beta2, alpha3beta4).
  द्वारा: Pym, L, और अन्य
  प्रकाशित: (2007)
• Subtype-specific actions of β-amyloid peptides on recombinant human neuronal nicotinic acetylcholine receptors (α7, α4β2, α3β4) expressed in Xenopus laevis oocytes
  द्वारा: Pym, L, और अन्य
  प्रकाशित: (2005)
• Alternative splicing of the Anopheles gambiae nicotinic acetylcholine receptor, Agamalphabeta9, generates both alpha and beta subunits.
  द्वारा: Jones, A, और अन्य
  प्रकाशित: (2009)
• Molecular determinants for competitive inhibition of alpha4beta2 nicotinic acetylcholine receptors.
  द्वारा: Iturriaga-Vásquez, P, और अन्य
  प्रकाशित: (2010)
• Additional acetylcholine (ACh) binding site at alpha4/alpha4 interface of (alpha4beta2)2alpha4 nicotinic receptor influences agonist sensitivity.
  द्वारा: Mazzaferro, S, और अन्य
  प्रकाशित: (2011)