Skip to content
VuFind
English
Deutsch
Español
Français
Italiano
日本語
Nederlands
Português
Português (Brasil)
中文(简体)
中文(繁體)
Türkçe
עברית
Gaeilge
Cymraeg
Ελληνικά
Català
Euskara
Русский
Čeština
Suomi
Svenska
polski
Dansk
slovenščina
اللغة العربية
বাংলা
Galego
Tiếng Việt
Hrvatski
हिंदी
Հայերէն
Українська
Sámegiella
Монгол
Language
All Fields
Title
Author
Subject
Call Number
ISBN/ISSN
Tag
Find
Advanced
GTPCH I overexpression decreas...
Cite this
Text this
Email this
Print
Export Record
Export to RefWorks
Export to EndNoteWeb
Export to EndNote
Permanent link
GTPCH I overexpression decreases atherosclerotic lesion formation in apolipoprotein E-deficient/eNOS transgenic mice
Bibliographic Details
Main Authors:
Takaya, T
,
Kawashima, S
,
Yamashita, T
,
Shinohara, M
,
Inoue, N
,
Hirata, K
,
Channon, K
,
Yokoyama, M
Format:
Conference item
Published:
2004
Holdings
Description
Similar Items
Staff View
Similar Items
Increased GTP-cyclohydrolase I expression but not vitamin C treatment restored accelerated atherosclerotic lesion formation in apolipoprotein E-deficient mice overexpressing endothelial nitric oxide synthase
by: Takaya, T, et al.
Published: (2004)
Increased endothelial tetrahydrobiopterin levels in GTPCH-transgenic mice
by: Alp, N, et al.
Published: (2002)
Stoichiometric relationships between endothelial tetrahydrobiopterin, endothelial NO synthase (eNOS) activity, and eNOS coupling in vivo: insights from transgenic mice with endothelial-targeted GTP cyclohydrolase 1 and eNOS overexpression.
by: Bendall, J, et al.
Published: (2005)
Endothelial-targeted GTP cyclohydrolase I overexpression increases endothelial tetrahydrobiopterin levels and preserves eNOS coupling in eNOS overexpressing mice
by: Bendall, J, et al.
Published: (2004)
A specific role for eNOS-derived reactive oxygen species in atherosclerosis progression.
by: Takaya, T, et al.
Published: (2007)