PRESERVATION OF ENDOTHELIAL GTP CYCLOHYDROLASE I ACTIVITY AND IMPROVEMENT OF RENAL NITRIC OXIDE AVAILABILITY PREVENT THE DEVELOPMENT OF DIABETIC NEPHROPATHY

Geahča maid

• Maintenance of endothelial guanosine triphosphate cyclohydrolase I ameliorates diabetic nephropathy.
  Dahkki: Kidokoro, K, et al.
  Almmustuhtton: (2013)
• Nitric oxide Bioavailability is preserved in diabetes by increased endothelial tetrahydrobiopterin synthesis in targeted GTP cyclohydrolase transgenic mice
  Dahkki: Alp, N, et al.
  Almmustuhtton: (2003)
• Tetrahydrobiopterin-dependent preservation of nitric oxide-mediated endothelial function in diabetes by targeted transgenic GTP-cyclohydrolase I overexpression.
  Dahkki: Alp, N, et al.
  Almmustuhtton: (2003)
• Tetrahydrobiopterin-dependent preservation of nitric oxide-mediated endothelial function in diabetes by targeted transgenic GTP-cyclohydrolase I over-expression
  Dahkki: Alp, N, et al.
  Almmustuhtton: (2003)
• Endothelial dysfunction promotes the transition from compensatory renal hypertrophy to kidney injury after unilateral nephrectomy in mice.
  Dahkki: Nagasu, H, et al.
  Almmustuhtton: (2012)