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Analysis of Lyn/CD22 double-deficient B cells in vivo demonstrates Lyn- and CD22-independent pathways affecting BCR regulation and B cell survival (vol 35, pg 3655, 2005)

Analysis of Lyn/CD22 double-deficient B cells in vivo demonstrates Lyn- and CD22-independent pathways affecting BCR regulation and B cell survival (vol 35, pg 3655, 2005)

Xehetasun bibliografikoak
Egile Nagusiak: Ferry, H, Crockford, T, Silver, K, Rust, N, Goodnow, C, Cornall, R
Formatua: Journal article
Argitaratua: 2005
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Antzeko izenburuak

  • Analysis of Lyn/CD22 double-deficient B cells in vivo demonstrates Lyn- and CD22-independent pathways affecting BCR regulation and B cell survival.
    nork: Ferry, H, et al.
    Argitaratua: (2005)
  • Erratum: Analysis of Lyn/CD22 double-deficient B cells in vivo demonstrates Lyn- and CD22- independent pathways affecting BCR regulation and B cell survival (European Journal of Immunology (2005) vol. 35 (12) (3714) 10.1002/eji.200535247)
    nork: Ferry, H, et al.
    Argitaratua: (2005)
  • Regulation of B cell antigen receptor signaling by the Lyn/CD22/SHP1 pathway.
    nork: Cornall, R, et al.
    Argitaratua: (1999)
  • Polygenic autoimmune traits: Lyn, CD22, and SHP-1 are limiting elements of a biochemical pathway regulating BCR signaling and selection.
    nork: Cornall, R, et al.
    Argitaratua: (1998)
  • Spontaneous class switching and B cell hyperactivity increase autoimmunity against intracellular self antigen in Lyn-deficient mice.
    nork: Silver, K, et al.
    Argitaratua: (2006)

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