Sharp injury to articular cartilage activates JNK and NF kappa B and induces expression of inflammatory response genes

Sharp injury to articular cartilage activates JNK and NF kappa B and induces expression of inflammatory response genes

מידע ביבליוגרפי
Main Authors:	Watt, F, Didangelos, A, Sawaji, Y, Saklatvala, J
פורמט:	Conference item
יצא לאור:	2006

פריטים דומים

Activin A is an anticatabolic autocrine cytokine in articular cartilage whose production is controlled by fibroblast growth factor 2 and NF-kappaB.
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Activin is an anti-catabolic cytokine produced by injured cartilage via release of fibroblast growth factor-2 and activation of nuclear factor kappa B
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Src and fibroblast growth factor 2 independently regulate signaling and gene expression induced by experimental injury to intact articular cartilage.
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Activin A is an anti-catabolic cytokine produced by injured cartilage via release of fibroblast growth factor-2 and activation of nuclear factor kappa B
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Anti-catabolic effect of fibroblast growth factor-2 in human articular cartilage
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INTERLEUKIN 1 INDUCES AGGRECAN DEGRADATION BY HUMAN ARTICULAR CHONDROCYTES THROUGH A TAK1/MKK4/JNK2 PATHWAY WITHOUT INVOLVEMENT OF NFKB
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Amygdalin Delays Cartilage Endplate Degeneration and Improves Intervertebral Disc Degeneration by Inhibiting NF-κB Signaling Pathway and Inflammatory Response [Corrigendum]
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