GTP cyclohydrolase I deficiency results in oxidative stress and pulmonary hypertension

GTP cyclohydrolase I deficiency results in oxidative stress and pulmonary hypertension

Номзүйн дэлгэрэнгүй
Үндсэн зохиолчид:	Khoo, J, Zhao, L, Alp, N, Bendall, J, Nicoll, T, Wilkins, MR, Channon, K
Формат:	Conference item
Хэвлэсэн:	2004

Ижил төстэй зүйлс

Endothelial-targeted GTP cyclohydrolase I overexpression increases endothelial tetrahydrobiopterin levels and preserves eNOS coupling in eNOS overexpressing mice
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GTP cyclohydrolase I expression, protein, and activity determine intracellular tetrahydrobiopterin levels, independent of GTP cyclohydrolase feedback regulatory protein expression.
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Cell-autonomous role of endothelial GTP cyclohydrolase 1 and tetrahydrobiopterin in blood pressure regulation.
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Cell-autonomous role of endothelial GTP cyclohydrolase 1 and tetrahydrobiopterin in blood pressure regulation
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Nitric oxide Bioavailability is preserved in diabetes by increased endothelial tetrahydrobiopterin synthesis in targeted GTP cyclohydrolase transgenic mice
-н: Alp, N, зэрэг
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Nitric oxide bioactivity in ApoE -/- mice is maintained by increased endothelial tetrahydrobiopterin synthesis via targeted GTP cyclohydrolase-1 over-expression
-н: Alp, N, зэрэг
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Tetrahydrobiopterin deficiency uncouples nitric oxide synthase and causes pulmonary hypertension
-н: Khoo, J, зэрэг
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303A cell-Autonomous role for endothelial GTP cyclohydrolase 1 and tetrahydrobiopterin in blood pressure regulation.
-н: Chuaiphichai, S, зэрэг
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Endothelial-targeted GTP cyclohydrolase I overexpression prevents blood pressure progression in mineralocorticoid hypertension
-н: Du, Y, зэрэг
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Endothelial-targeted GTP cyclohydrolase I overexpression prevents blood pressure progression in mineralocorticoid hypertension\
-н: Du, Y, зэрэг
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Endothelial-specific GTP cyclohydrolase I overexpression prevents blood pressure progression in salt-sensitive hypertension
-н: Chen, A, зэрэг
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Cardiomyocyte-targeted overexpression of GTP cyclohydrolase-1 increases nNOS activity and hastens myocardial relaxation
-н: Carnicer, R, зэрэг
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Relationships between nitric oxide-mediated endothelial function, eNOS coupling and blood pressure revealed by eNOS-GTP cyclohydrolase 1 double transgenic mice.
-н: Adlam, D, зэрэг
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Tetrahydrobiopterin-dependent preservation of nitric oxide-mediated endothelial function in diabetes by targeted transgenic GTP-cyclohydrolase I overexpression.
-н: Alp, N, зэрэг
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Endothelial-specific GTP cyclohydrolase overexpression reduces blood pressure in salt-sensitive hypertension
-н: Chen, A, зэрэг
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Parkinson's disease in GTP cyclohydrolase 1 mutation carriers.
-н: Ryan, B, зэрэг
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Cardiomyocyte GTP cyclohydrolase 1 and tetrahydrobiopterin increase NOS1 activity and accelerate myocardial relaxation
-н: Carnicer, R, зэрэг
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Cardiomyocyte GTP cyclohydrolase 1 and tetrahydrobiopterin increase NOS1 activity and accelerate myocardial relaxation
-н: Carnicer, R, зэрэг
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Endothelium-specific GTP cyclohydrolase I overexpression attenuates blood pressure progression in salt-sensitive low-renin hypertension.
-н: Du, Y, зэрэг
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Tetrahydrobiopterin deficiency causes pulmonary hypertension: Effects on lung superoxide levels, hypoxic pulmonary vasoconstriction and vascular remodelling
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GTP cyclohydrolase II structure and mechanism.
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GTP Cyclohydrolase/BH4 Pathway Protects EPC Function via Suppressing Oxidative Stress and Thrombospondin-1 in Salt-Sensitive Hypertension
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GTP cyclohydrolase I/BH4 pathway protects EPCs via suppressing oxidative stress and thrombospondin-1 in salt-sensitive hypertension
-н: Xie, H, зэрэг
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GTP cyclohydrolase I/BH4 pathway protects EPCs via suppressing oxidative stress and thrombospondin-1 in salt-sensitive hypertension.
-н: Xie, H, зэрэг
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Decreased Cardiac Allograft Rejection in Mice Over-Expressing Human GTP Cyclohydrolase I.
-н: Irina, I, зэрэг
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Endothelium-specific GTP cyclohydrolase I overexpression accelerates refractory wound healing by suppressing oxidative stress in diabetes.
-н: Tie, L, зэрэг
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Tetrahydrobiopterin-dependent preservation of nitric oxide-mediated endothelial function in diabetes by targeted transgenic GTP-cyclohydrolase I over-expression
-н: Alp, N, зэрэг
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Increased endothelial tetrahydrobiopterin synthesis by targeted transgenic GTP-cyclohydrolase I overexpression reduces endothelial dysfunction and atherosclerosis in ApoE-knockout mice.
-н: Alp, N, зэрэг
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Cardiomyocyte-Targeted Overexpression of GTP Cyclohydrolase-1 Increases Myocyte Lusitropy and Reduces Superoxide Production
-н: Carnicer, R, зэрэг
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Endothelium-targeted GTP cyclohydrolase I overexpression inhibits wire injury-induced restenosis in mouse carotid artery
-н: Chen, A, зэрэг
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Endotoxemia-related acute kidney injury in transgenic mice with endothelial overexpression of GTP cyclohydrolase-1.
-н: Wang, W, зэрэг
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Systemic adenovirus-mediated gene transfer of human GTP cyclohydrolase-1 increases liver, plasma and aortic tetrahydrobiopterin levels in mice
-н: Alp, N, зэрэг
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GTP cyclohydrolase I prevents diabetic-impaired endothelial progenitor cells and wound healing by suppressing oxidative stress/thrombospondin-1.
-н: Tie, L, зэрэг
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GTP cyclohydrolase as a novel mechanism for tumour growth
-н: Chen, L, зэрэг
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Assay for GTP Cyclohydrolase II Activity in Bacterial Extracts
-н: Svetlana Yurgel, зэрэг
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Prevention of hypoxia-induced pulmonary hypertension and vascular remodelling by targeting endothelial tetrahydrobiopterin-dependent nitric oxide synthase regulation
-н: Khoo, J, зэрэг
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GTP cyclohydrolase I gene transfer augments intracellular tetrahydrobiopterin in human endothelial cells: effects on nitric oxide synthase activity, protein levels and dimerisation.
-н: Cai, S, зэрэг
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Pivotal role for endothelial tetrahydrobiopterin in pulmonary hypertension.
-н: Khoo, J, зэрэг
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GTP cyclohydrolase I gene transfer reverses tetrahydrobiopterin deficiency and increases nitric oxide synthesis in endothelial cells and isolated vessels from diabetic rats.
-н: Meininger, C, зэрэг
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Cardiac myocyte-specific overexpression of human GTP cyclohydrolase I protects against acute cardiac allograft rejection.
-н: Ionova, I, зэрэг
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