The cardiac sympathetic co-transmitter galanin reduces acetylcholine release and vagal bradycardia: implications for neural control of cardiac excitability.
The autonomic phenotype of congestive cardiac failure is characterised by high sympathetic drive and impaired vagal tone, which are independent predictors of mortality. We hypothesize that impaired bradycardia to peripheral vagal stimulation following high-level sympathetic drive is due to sympatho-...
मुख्य लेखकों: | Herring, N, Cranley, J, Lokale, M, Li, D, Shanks, J, Alston, E, Girard, B, Carter, E, Parsons, R, Habecker, B, Paterson, D |
---|---|
स्वरूप: | Journal article |
भाषा: | English |
प्रकाशित: |
Elsevier
2012
|
समान संसाधन
-
The cardiac sympathetic co-transmitter galanin reduces acetylcholine release and vagal bradycardia: Implications for neural control of cardiac excitability
द्वारा: Herring, N, और अन्य
प्रकाशित: (2011) -
Galanin reduces cardiac vagal acetylcholine release and bradycardia via a GalR1, protein kinase C dependent pathway
द्वारा: Herring, N, और अन्य
प्रकाशित: (2010) -
Neuropeptide Y reduces acetylcholine release and vagal bradycardia via a Y2 receptor-mediated, protein kinase C-dependent pathway.
द्वारा: Herring, N, और अन्य
प्रकाशित: (2008) -
Natriuretic peptides like NO facilitate cardiac vagal neurotransmission and bradycardia via a cGMP pathway.
द्वारा: Herring, N, और अन्य
प्रकाशित: (2001) -
Nitric oxide-cGMP pathway facilitates acetylcholine release and bradycardia during vagal nerve stimulation in the guinea-pig in vitro.
द्वारा: Herring, N, और अन्य
प्रकाशित: (2001)