Direct induction by melanoma, prostate cancer and Ewing's sarcoma tumour cells of osteoclast formation by a RANKL-independent mechanism

Direct induction by melanoma, prostate cancer and Ewing's sarcoma tumour cells of osteoclast formation by a RANKL-independent mechanism

Xehetasun bibliografikoak
Egile Nagusiak:	Lau, Y, Sabokbar, A, Giele, H, Gibbons, C, Cerundolo, V, Athanasou, N
Formatua:	Conference item
Argitaratua:	2005

Antzeko izenburuak

Cellular and humoral mechanisms of osteoclast formation in Ewing's sarcoma.
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Argitaratua: (2007)

Ewing sarcoma cells express RANKL and support osteoclastogenesis
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Argitaratua: (2011)

Ewing sarcoma cells express RANKL and support osteoclastogenesis.
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Argitaratua: (2011)

Ewing's sarcoma cells promote osteoclast formation
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Rankl-independent induction of human osteoclast formation and bone resorption by TGF-beta
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RANKL-dependent and RANKL-independent mechanisms of macrophage-osteoclast differentiation in breast cancer.
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Bone stromal cells in pagetic bone and Paget's sarcoma express RANKL and support human osteoclast formation.
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Argitaratua: (2006)

Involvement of RANKL-dependent/independent mechanisms in osteoclast formation in aseptic loosening
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Sarcoma-associated macrophages differentiate into osteoclastic bone-resorbing cells
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Localized Ewing sarcoma of the tibia.
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IL-6 induces human osteoclast formation and bone resorption independent of RANK/RANKL interaction
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Interleukin-6 and interleukin-11 support human osteoclast formation by a RANKL-independent mechanism.
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Human osteoclast formation and bone resorption by a mechanism independent of RANK/RANKL: Role of IL-6 and IL-11.
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Transforming growth factor-beta induces osteoclast formation in the absence of RANKL.
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Heterogeneity in Ewing sarcoma
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RANKL and M-CSF are required for osteoclast differentiation by macrophages in breast cancer
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RANKL-independent human osteoclast formation with APRIL, BAFF, NGF, IGF I and IGF II.
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RANKL-independent human osteoclast formation with APRIL, BAFF, NGF, IGF I and IGF II.
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Osteoclast differentiation in Paget's disease is hypersensitive to RANKL.
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Metal wear-induced pseudotumour following an endoprosthetic knee replacement for Ewing sarcoma
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Malignant melanoma and bone resorption.
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Human mesenchymal tumour-associated macrophages differentiate into osteoclastic bone-resorbing cells.
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RANKL-independent osteoclastogenesis in Paget's disease
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Targeted treatment of Ewing sarcomas: Current status
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RANKL plays an important role as activator of osteoclastic bone resorption in acute Charcot osteoarthropathy
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Human tumour associated macrophages are capable of osteoclastic differentiation and bone resorption
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Hypoxia and hypoglycaemia in Ewing's sarcoma and osteosarcoma: regulation and phenotypic effects of Hypoxia-Inducible Factor.
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Proinflammatory cytokine (TNFalpha/IL-1alpha) induction of human osteoclast formation.
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Secreted frizzled-related protein inhibits TNFalpha- or RANKL-dependent osteoclast formation
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Secreted frizzled-related protein-1 inhibits RANKL-dependent osteoclast formation.
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Combinatorial drug screening identifies Ewing sarcoma-specific sensitivities
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Effect of corticosteroids on human osteoclast formation and activity.
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The effect of corticosteroids on human osteoclast formation and activity
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Humoral factors act directly on circulating osteoclast precursors to control osteoclast formation
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Formation and isolation of human osteoclasts
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Malignant neck mass in disguise - atypical presentation of extraskeletal Ewing's sarcoma
nork: Poh, Nuan Leng, et al.
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LINGO-1 is a New Therapy Target and Biomarker for Ewing Sarcoma
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Osteoclast formation in Gorham-Stout disease
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Subungual melanoma with osteocartilaginous differentiation.
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Osteoclast-like cells in soft tissue leiomyosarcomas.
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