Regulation of myocyte function by gch1 overexpression mediated increase in nitric oxide

Regulation of myocyte function by gch1 overexpression mediated increase in nitric oxide

التفاصيل البيبلوغرافية
المؤلفون الرئيسيون:	Hijazo, R, Hale, AB, Liu, X, Suffredini, S, Bendall, J, Lim, G, Alp, N, Channon, K, Casadei, B
التنسيق:	Conference item
منشور في:	2012

مواد مشابهة

Cardiomyocyte-Targeted Overexpression of GTP Cyclohydrolase-1 Increases Myocyte Lusitropy and Reduces Superoxide Production
حسب: Carnicer, R, وآخرون
منشور في: (2010)

Cardiomyocyte-targeted overexpression of GTP cyclohydrolase-1 increases nNOS activity and hastens myocardial relaxation
حسب: Carnicer, R, وآخرون
منشور في: (2011)

A requirement for Gch1 and tetrahydrobiopterin in embryonic development.
حسب: Douglas, G, وآخرون
منشور في: (2015)

EPR quantification of vascular nitric oxide production in genetically modified mouse models.
حسب: Khoo, J, وآخرون
منشور في: (2004)

Cardiomyocyte GTP cyclohydrolase 1 and tetrahydrobiopterin increase NOS1 activity and accelerate myocardial relaxation
حسب: Carnicer, R, وآخرون
منشور في: (2012)

Enhancement of beta adrenergic responsiveness in myocytes lacking nitric oxide synthase 1
حسب: Ashley, E, وآخرون
منشور في: (2001)

Conditional knockout of GCH1 in leukocytes and endothelial cells: A novel model to study tetrahydrobiopterin-dependent NOS functions in vivo
حسب: McNeill, E, وآخرون
منشور في: (2012)

Nitric oxide bioactivity in ApoE -/- mice is maintained by increased endothelial tetrahydrobiopterin synthesis via targeted GTP cyclohydrolase-1 over-expression
حسب: Alp, N, وآخرون
منشور في: (2003)

Tetrahydrobiopterin-dependent preservation of nitric oxide-mediated endothelial function in diabetes by targeted transgenic GTP-cyclohydrolase I overexpression.
حسب: Alp, N, وآخرون
منشور في: (2003)

Cardiomyocyte GTP cyclohydrolase 1 and tetrahydrobiopterin increase NOS1 activity and accelerate myocardial relaxation
حسب: Carnicer, R, وآخرون
منشور في: (2012)

Endothelial-targeted GTP cyclohydrolase I overexpression increases endothelial tetrahydrobiopterin levels and preserves eNOS coupling in eNOS overexpressing mice
حسب: Bendall, J, وآخرون
منشور في: (2004)

Molecular mechanisms underlying the myocardial nitric oxide-redox imbalance in human atrial fibrillation
حسب: Reilly, SN, وآخرون
منشور في: (2009)

Cardiac nitric oxide synthase 1 regulates basal and beta-adrenergic contractility in murine ventricular myocytes.
حسب: Ashley, E, وآخرون
منشور في: (2002)

Dihydrofolate reductase is required to maintain endothelial nitric oxide synthase coupling in vivo: Insights from methotrexate-treated BH4 deficient and GTPCH overexpressing mice
حسب: Crabtree, M, وآخرون
منشور في: (2010)

Endothelial-specific Nox2 overexpression increases vascular superoxide and macrophage recruitment in ApoE⁻/⁻ mice.
حسب: Douglas, G, وآخرون
منشور في: (2012)

Endogenous nitric oxide formation in cardiac myocytes does not control respiration during β‐adrenergic stimulation
حسب: Kohlhaas, M, وآخرون
منشور في: (2017)

Roles for endothelial cell and macrophage Gch1 and tetrahydrobiopterin in atherosclerosis progression
حسب: Douglas, G, وآخرون
منشور في: (2018)

Regulation of mycobacterial infection by macrophage Gch1 and tetrahydrobiopterin
حسب: McNeill, E, وآخرون
منشور في: (2018)

Radiochemical HPLC detection of arginine metabolism: measurement of nitric oxide synthesis and arginase activity in vascular tissue.
حسب: de Bono, J, وآخرون
منشور في: (2007)

Does nitric oxide regulate the hyperpolarisation-activated inward current in ventricular myocytes from spontaneously hypertensive rats?
حسب: Bryant, S, وآخرون
منشور في: (2000)

Does nitric oxide regulate the hyperpolarisation-activated inward current in ventricular myocytes from spontaneously hypertensive rats?
حسب: Sears, C, وآخرون
منشور في: (2000)

Regulation of mycobacterial infection by macrophage Gch1 and tetrahydrobiopterin
حسب: Eileen McNeill, وآخرون
منشور في: (2018-12-01)

Rapid, direct effects of statin treatment on arterial redox state and nitric oxide bioavailability in human atherosclerosis via tetrahydrobiopterin-mediated endothelial nitric oxide synthase coupling.
حسب: Antoniades, C, وآخرون
منشور في: (2011)

Rapid, direct effects of statin treatment on arterial redox state and nitric oxide bioavailability in human atherosclerosis via tetrahydrobiopterin- mediated endothelial nitric oxide synthase coupling
حسب: Antoniades, C, وآخرون
منشور في: (2011)

Tetrahydrobiopterin deficiency uncouples nitric oxide synthase and causes pulmonary hypertension
حسب: Khoo, J, وآخرون
منشور في: (2004)

The subcellular localisation of neuronal nitric oxide synthase determines the downstream effects of NO on myocardial function
حسب: Carnicer, R, وآخرون
منشور في: (2017)

Nitric oxide does not modulate the hyperpolarization-activated current, I(f), in ventricular myocytes from spontaneously hypertensive rats.
حسب: Bryant, S, وآخرون
منشور في: (2001)

201 Regulation of INOS Function, Redox State and NRF2 Activation by Macrophage GCH1 and Tetrahydrobiopterin.
حسب: McNeill, E, وآخرون
منشور في: (2014)

A cardiac neuronal nitric oxide synthase isoform regulates contraction and calcium handling in murine ventricular myocytes
حسب: Sears, C, وآخرون
منشور في: (2002)

A cardiac neuronal nitric oxide synthase isoform regulates contraction and calcium handling in murine ventricular myocytes
حسب: Sears, C, وآخرون
منشور في: (2002)

Hydrogen peroxide differentially modulates cardiac myocyte nitric oxide synthesis
حسب: Sartoretto, Juliano, وآخرون
منشور في: (2012)

Regulation of endothelial nitric oxide synthase by tetrahydrobiopterin in vascular disease.
حسب: Alp, N, وآخرون
منشور في: (2004)

Dihydrofolate reductase protects endothelial nitric oxide synthase from uncoupling in tetrahydrobiopterin deficiency.
حسب: Crabtree, M, وآخرون
منشور في: (2011)

Dihydrofolate reductase protects endothelial nitric oxide synthase from uncoupling in tetrahydrobiopterin deficiency
حسب: Crabtree, M, وآخرون
منشور في: (2011)

Neuronal nitric oxide synthase (NOS1) regulates beta-adrenergic contraction and calcium handling in isolated murine ventricular myocytes
حسب: Zhang, Y, وآخرون
منشور في: (2003)

Reduced nitric oxide bioactivity increases platelet activation in mice with streptozotocin-induced diabetes mellitus
حسب: Schafer, A, وآخرون
منشور في: (2003)

Rapid regulation of platelet activation in vivo by nitric oxide
حسب: Schafer, A, وآخرون
منشور في: (2003)

Critical role for tetrahydrobiopterin recycling by dihydrofolate reductase in regulation of endothelial nitric-oxide synthase coupling: relative importance of the de novo biopterin synthesis versus salvage pathways.
حسب: Crabtree, M, وآخرون
منشور في: (2009)

Regulation of β-adrenergic control of heart rate by GTP-cyclohydrolase 1 (GCH1) and tetrahydrobiopterin.
حسب: Adlam, D, وآخرون
منشور في: (2012)

Electrophysiological and mechanical properties of ventricular myocytes isolated from wild-type mice and mice with disruption of the neuronal nitric oxide synthase gene
حسب: Sears, C, وآخرون
منشور في: (2001)