Increased NAD(P)H oxidase activity in coronary arteries from patients with coronary artery disease

Increased NAD(P)H oxidase activity in coronary arteries from patients with coronary artery disease

Xehetasun bibliografikoak
Egile Nagusiak:	Guzik, T, Kapelak, B, Garlicki, M, Wierzbicki, K, Guzik, B, Dziatkowiak, A, Korbut, R, Channon, K
Formatua:	Journal article
Argitaratua:	2001

Antzeko izenburuak

Mechanisms of increased vascular superoxide generation in human coronary arteries from patients with coronary artery disease
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Systemic regulation of vascular NAD(P)H oxidase activity and nox isoform expression in human arteries and veins
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Vascular superoxide production in human coronary artery disease. Relationships between Nox expression and oxidase activity
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Systemic regulation of vascular NAD(P)H oxidase activity and nox isoform expression in human arteries and veins.
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Vascular superoxide generation by NAD(P)H oxidase is increased in humans with type II diabetes mellitus
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Atrial NAD(P)H oxidase activity predicts the development of atrial fibrillation after on-pump coronary artery bypass graft surgery
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Systemic upregulation of vascular NAD(P)H oxidase in human atherosclerosis: Association with clinical risk factors.
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Relationship of NAD(P)H oxidase activity in human blood vessels to endothelial dysfunction and clinical risk factors
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Functional impact of genetic polymorphisms in NAD(P)H oxidase p22phox subunit on vascular superoxide production in atherosclerosis
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A myocardial nox2 containing NAD(P)H oxidase contributes to oxidative stress in human atrial fibrillation (AF)
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A myocardial Nox2 containing NAD(P)H oxidase contributes to oxidative stress in human atrial fibrillation.
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Functional effect of the C242T polymorphism in the NAD(P)H oxidase p22phox gene on vascular superoxide production in atherosclerosis.
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Functional importance of C242T polymorphism in NAD(P)H oxidase p22phox subunit on vascular superoxide production in atherosclerosis
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UltraRapid communications : vascular superoxide production by NAD(P)H OxidaseAssociation with endothelial dysfunction and clinical risk factors
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Protein kinase C mediates increased vascular oxidative in stress in patients with type II diabetes mellitus
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Association of superoxide production by NAD(P)H oxidase with systaemic endothelial dysfunction in human blood vessels: relationship to clinical risk factors
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