High-density lipoproteins retard the progression of atherosclerosis and favorably remodel lesions without suppressing indices of inflammation or oxidation.

Παρόμοια τεκμήρια

• Elevating high-density lipoprotein cholesterol in apolipoprotein E-deficient mice remodels advanced atherosclerotic lesions by decreasing macrophage and increasing smooth muscle cell content.
  ανά: Rong, J, κ.ά.
  Έκδοση: (2001)
• Human apolipoprotein A-I transgene expression retards the progression of atherosclerosis in apolipoprotein E deficient mice fed Western-type diet (WD)
  ανά: Choudhury, R, κ.ά.
  Έκδοση: (2001)
• Apolipoprotein AI overexpression remodels large, complex atherosclerotic lesions to have characteristics of more stable plaques in apoE-deficient mice
  ανά: Rong, J, κ.ά.
  Έκδοση: (2001)
• Immuno-laser capture microdissection (LCM) enriches macrophage foam cell RNA for the molecular analysis of atherosclerotic lesions
  ανά: Trogan, E, κ.ά.
  Έκδοση: (2002)
• Laser capture microdissection analysis of gene expression in macrophages from atherosclerotic lesions of apolipoprotein E-deficient mice.
  ανά: Trogan, E, κ.ά.
  Έκδοση: (2002)