Systemic and Local Renal Complement Activation in Brain-Dead Donors

Systemic and Local Renal Complement Activation in Brain-Dead Donors

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Bibliografiska uppgifter
Huvudupphovsmän:	Damman, J, Leuvenink, H, Nijboer, W, van Oeveren, W, Ploeg, R, Seelen, M
Materialtyp:	Conference item
Publicerad:	2009

Liknande verk

SYSTEMIC AND LOCAL RENAL COMPLEMENT ACTIVATION IN BRAIN-DEAD DONORS
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Renal Upregulation of Complement C3 and Fibrinogen as Part of the Acute Phase Response in Brain-Dead Donors
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Local renal upregulation of complement C3 and fibrinogen as part of the acute phase response in brain-dead donors
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Targeting complement activation in brain-dead donors improves renal function after transplantation.
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Local renal complement C3 induction by donor brain death is associated with reduced renal allograft function after transplantation.
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Crosstalk between complement and Toll-like receptor activation in relation to donor brain death and renal ischemia-reperfusion injury.
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Complement and renal transplantation: from donor to recipient.
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Crosstalk between Complement and Toll-like Receptor Activation in Relation to Donor Brain Death and Renal Ischemia-Reperfusion Injury (vol 11, pg 660, 2011)
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ANGIOPOIETIN IN LIVING AND DECEASED BRAIN DEAD KIDNEY DONORS
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Systemic complement activation in deceased donors is associated with acute rejection after renal transplantation in the recipient.
av: Damman, J, et al.
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Systemic Complement Activation in Deceased Donors Is Associated with Acute Allograft Rejection after Renal Transplantation.
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Association of complement C3 gene variants with renal transplant outcome of deceased cardiac dead donor kidneys
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Association of complement C3 gene variants with renal transplant outcome of deceased cardiac dead donor kidneys.
av: Damman, J, et al.
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Erratum: Crosstalk between complement and toll-like receptor activation in relation to donor brain death and renal ischemia-reperfusion injury (American Journal of Transplantation (2011) 11 (660-669) DOI: 10.1111/j.1600-6143.2011. 03475.x)
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Increased intestinal permeability in the brain dead donor
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ACTIVATION OF HEMOSTASIS IN BRAIN-DEAD ORGAN DONORS
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Inflammatory Angiopoietin Response in Deceased Brain Dead Donors
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Dysfunction of Blood-Brain Barrier in Deceased Brain Dead Donors Leads to Increased Serum IL-6 and a Decreased Outcome after Transplantation.
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Activation of hemostasis in brain dead organ donors: an observational study.
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Intestinal permeability and inflammatory balance reflected by LPS binding protein and Angiopoietin 1 and 2, in brain dead and living donors
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Kidney grafts from brain dead donors: Inferior quality or opportunity for improvement?
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Donor pretreatment with carbamylated erythropoietin in a brain death model reduces inflammation more effectively than erythropoietin while preserving renal function.
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Kidney injury molecule-1 is an early noninvasive indicator for donor brain death-induced injury prior to kidney transplantation.
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Cumulative negative effects of donor brain death and subsequent ischemia/reperfusion on kidney graft injury and function.
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Cumulative negative effects of donor brain death and subsequent ischemia/reperfusion on kidney graft injury and function
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BIOCHEMICAL ASSESSMENT OF MACHINE-PERFUSED DONOR KIDNEYS
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Proteomic analysis of machine perfusion solution from brain dead donor kidneys reveals that elevated complement, cytoskeleton and lipid metabolism proteins are associated with 1-year outcome.
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Effect of brain death and donor treatment on organ inflammatory response and donor organ viability
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Effects of brain death on stress and inflammatory response in the human donor kidney.
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Effect of brain death on gene expression and tissue activation in human donor kidneys.
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Identification of differentially expressed genes in kidneys from brain dead donors using oligonucleotide arrays
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Lectin complement pathway gene profile of the donor and recipient does not influence graft outcome after kidney transplantation
av: Damman, J, et al.
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Lectin complement pathway gene profile of the donor and recipient does not influence graft outcome after kidney transplantation.
av: Damman, J, et al.
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Complement Therapeutics in the Multi-Organ Donor: Do or Don't?
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Early events in kidney donation: progression of endothelial activation, oxidative stress and tubular injury after brain death.
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How important is the duration of the brain death period for the outcome in kidney transplantation?
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HYPOTHERMIC IN SITU MACHINE PERFUSION OF DONOR ORGANS DURING NON-HEART-BEATING DONATION
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Brain death induced renal injury.
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