Atorvastatin rapidly reduces myocardial superoxide and peroxynitrite generation in human atherosclerosis, by suppressing NADPH-oxidase activity independently of LDL lowering

מידע ביבליוגרפי
Main Authors:	Antoniades, C, Demosthenous, M, Reilly, S, Margaritis, M, Jayaram, R, Nahar, K, Tousoulis, D, Stefanadis, C, Channon, K, Casadei, B
פורמט:	Journal article
יצא לאור:	2011

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Preoperative atorvastatin treatment rapidly improves vein graft redox state by inhibiting rac1 and NADPH oxidase activity in patients undergoing elective CABG
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Letter regarding article by Becker et al, "Hyperhomocysteinemia, a cardiac metabolic disease: role of nitric oxide and the p22phox subunit of NADPH oxidase".
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Coupling of phagocytic NADPH oxidase activity and mitochondrial superoxide production
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Regulation of NADPH Oxidase-Mediated Superoxide Production by Acetylation and Deacetylation
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Nitric oxide modulates superoxide release and peroxynitrite formation in human blood vessels.
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Nitric oxide-superoxide interactions in human blood vessels: implications for vascular peroxynitrite formation
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Cyclic Hydroxylamines as Monitors of Peroxynitrite and Superoxide-Revisited
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The Interplay Between Perivascular Adipose Tissue and the Arterial Wall Regulates NADPH-Oxidase Activity in Human Vessels
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Genetic Variability of Adiponectin Gene Defines the Biosynthesis Rate of Adiponectin in Adipose Tissue and Regulates Superoxide Generation in Human Arteries in vivo
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Atorvastatin rapidly reduces myocardial superoxide and peroxynitrite generation in human atherosclerosis, by suppressing NADPH-oxidase activity independently of LDL lowering

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