Single point mutations in ATP synthase compensate for mitochondrial genome loss in trypanosomes

Viability of the tsetse fly-transmitted African trypanosome Trypanosoma brucei depends on maintenance and expression of its kinetoplast (kDNA), the mitochondrial genome of this parasite and a putative target for veterinary and human antitrypanosomatid drugs. However, the closely related animal patho...

詳細記述

書誌詳細
主要な著者: Dean, S, Gould, MK, Dewar, CE, Schnaufer, AC
フォーマット: Journal article
言語:English
出版事項: National Academy of Sciences 2013