Altered polarity and expression of H+-ATPase without ultrastructural changes in kidneys of Dent's disease patients.

Documenti analoghi

• Intra-renal and subcellular distribution of the human chloride channel, CLC-5, reveals a pathophysiological basis for Dent's disease.
  di: Devuyst, O, et al.
  Pubblicazione: (1999)
• Dent's disease.
  di: Devuyst, O, et al.
  Pubblicazione: (2010)
• Dent's disease
  di: Thakker Rajesh V, et al.
  Pubblicazione: (2010-10-01)
• Mice lacking renal chloride channel, CLC-5, are a model for Dent's disease, a nephrolithiasis disorder associated with defective receptor-mediated endocytosis.
  di: Wang, S, et al.
  Pubblicazione: (2000)
• OCRL1 mutations in Dent 2 patients suggest a mechanism for phenotypic variability.
  di: Shrimpton, A, et al.
  Pubblicazione: (2009)