IgG subclasses of disease-specific antibodies and complement activation in vitro in myasthenia gravis and neuromyelitis optica

IgG subclasses of disease-specific antibodies and complement activation in vitro in myasthenia gravis and neuromyelitis optica

Номзүйн дэлгэрэнгүй
Үндсэн зохиолчид:	Jacob, S, Leite, M, Jarius, S, Viegas, S, Cossins, J, Beeson, D, Morgan, B, Vincent, A
Формат:	Conference item
Хэвлэсэн:	2007

Ижил төстэй зүйлс

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NMO-IgG/Aqp4-Ab belongs to the IgG1 subclass and activates complement in vitro
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Frequency of Aqp4-Ab in patients with neuromyelitis optica (Devic's syndrome) is higher than predicted by NMO-IgG testing
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MuSK myasthenia gravis IgG4 disrupts the interaction of LRP4 with MuSK but both IgG4 and IgG1-3 can disperse preformed agrin-independent AChR clusters
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MuSK myasthenia gravis IgG4 disrupts the interaction of LRP4 with MuSK but both IgG4 and IgG1-3 can disperse preformed agrin-independent AChR clusters.
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Neuromyelitis optica IgG causes placental inflammation and fetal death in mice
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Myasthenia gravis seronegative for acetylcholine receptor antibodies.
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Aquaporin-4 antibodies in neuromyelitis optica and longitudinally extensive transverse myelitis.
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Sequential voltage-gated potassium channel and aquaporin-4 antibodies in a patient with seropositive myasthenia gravis, limbic encephalitis and neuromyelitis optica
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Seroprevalence of Neuromyelitis Optica-IgG of Childhood Compared to Adults
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Presence and pathogenic relevance of antibodies to clustered acetylcholine receptor in ocular and generalized myasthenia gravis.
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Complement-independent bystander injury in AQP4-IgG seropositive neuromyelitis optica produced by antibody-dependent cellular cytotoxicity
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Neuromyelitis optica in patients with gluten sensitivity associated with antibodies to aquaporin-4.
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Plasmablasts as migratory IgG-producing cells in the pathogenesis of neuromyelitis optica.
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Clinical fluctuations in MuSK myasthenia gravis are related to antigen-specific IgG4 instead of IgG1.
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