IgG subclasses of disease-specific antibodies and complement activation in vitro in myasthenia gravis and neuromyelitis optica

IgG subclasses of disease-specific antibodies and complement activation in vitro in myasthenia gravis and neuromyelitis optica

Bibliografische gegevens
Hoofdauteurs:	Jacob, S, Leite, M, Jarius, S, Viegas, S, Cossins, J, Beeson, D, Morgan, B, Vincent, A
Formaat:	Conference item
Gepubliceerd in:	2007

Gelijkaardige items

IgG subclasses of disease-specific antibodies and complement activation in-vitro in seronegative myasthenia gravis and neuromyelitis optica
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IgG1 antibodies to acetylcholine receptors in 'seronegative' myasthenia gravis.
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Myasthenia Gravis and Neuromyelitis Optica Association
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Immunopathogenesis in Myasthenia Gravis and Neuromyelitis Optica
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NMO-IgG in the diagnosis of neuromyelitis optica.
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NMO-IgG/Aqp4-Ab belongs to the IgG1 subclass and activates complement in vitro
door: Jarius, S, et al.
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Neuromyelitis optica and longitudinally extensive transverse myelitis following thymectomy for myasthenia gravis
door: Jarius, S, et al.
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Myasthenia gravis and neuromyelitis optica spectrum disorder: a multicenter study of 16 patients.
door: Leite, M, et al.
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Revised diagnostic criteria for neuromyelitis optica--incorporation of NMO-IgG status.
door: Jarius, S, et al.
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Frequency of Aqp4-Ab in patients with neuromyelitis optica (Devic's syndrome) is higher than predicted by NMO-IgG testing
door: Jarius, S, et al.
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Antibodies to AChRs in seronegative myasthenia gravis
door: Leite, M, et al.
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MuSK myasthenia gravis IgG4 disrupts the interaction of LRP4 with MuSK but both IgG4 and IgG1-3 can disperse preformed agrin-independent AChR clusters
door: Koneczny, I, et al.
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MuSK myasthenia gravis IgG4 disrupts the interaction of LRP4 with MuSK but both IgG4 and IgG1-3 can disperse preformed agrin-independent AChR clusters.
door: Koneczny, I, et al.
Gepubliceerd in: (2013)

MuSK myasthenia gravis IgG4 disrupts the interaction of LRP4 with MuSK but both IgG4 and IgG1-3 can disperse preformed agrin-independent AChR clusters.
door: Inga Koneczny, et al.
Gepubliceerd in: (2013-01-01)

Clinical and immunological correlates in myasthenia gravis associated with musk antibodies
door: Viegas, S, et al.
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Neuromyelitis optica IgG causes placental inflammation and fetal death.
door: Saadoun, S, et al.
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Devic's neuromyelitis optica: Prognostic implications of NMO IgG status in Turkish patients
door: Akman-Demir, G, et al.
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Complement-dependent bystander injury to neurons in AQP4-IgG seropositive neuromyelitis optica
door: Tianjiao Duan, et al.
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Neuromyelitis optica IgG causes placental inflammation and fetal death in mice
door: Saadoun, S, et al.
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An audit of the diagnostic usefulness of the NMO-IgG assay for neuromyelitis optica
door: Littleton, E, et al.
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Antibodies to clustered acetylcholine receptor and thymic changes in seronegative myasthenia gravis
door: Leite, M, et al.
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Role of complement in myasthenia gravis
door: Pyae Phyo San, et al.
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Inhibition of acetylcholine receptor function by seronegative myasthenia gravis non-IgG factor correlates with desensitisation.
door: Spreadbury, I, et al.
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Myasthenia gravis seronegative for acetylcholine receptor antibodies.
door: Vincent, A, et al.
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Aquaporin-4 antibodies in neuromyelitis optica and longitudinally extensive transverse myelitis.
door: Waters, P, et al.
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Sequential voltage-gated potassium channel and aquaporin-4 antibodies in a patient with seropositive myasthenia gravis, limbic encephalitis and neuromyelitis optica
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PATHOGENIC MECHANISMS UNDERLYING MYASTHENIA GRAVIS ASSOCIATED WITH MUSK ANTIBODIES
door: Viegas, S, et al.
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Seroprevalence of Neuromyelitis Optica-IgG of Childhood Compared to Adults
door: J Gordon Millichap
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Neuromyelitis optica-IgG testing in an Indian cohort with neuromyelitis optica and related demyelinating disorders: Our experience
door: Narayanan Unni, et al.
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Antibodies identified by cell-based assays in myasthenia gravis and associated diseases.
door: Vincent, A, et al.
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Inebilizumab treatment in a patient with co-occurring AQP4-IgG positive neuromyelitis optica spectrum disorder and myasthenia gravis: a case report and literature review
door: Xiaoqian Song, et al.
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Presence and pathogenic relevance of antibodies to clustered acetylcholine receptor in ocular and generalized myasthenia gravis.
door: Jacob, S, et al.
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Complement-independent bystander injury in AQP4-IgG seropositive neuromyelitis optica produced by antibody-dependent cellular cytotoxicity
door: Tianjiao Duan, et al.
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Neuromyelitis optica in patients with gluten sensitivity associated with antibodies to aquaporin-4.
door: Jarius, S, et al.
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Complement activation in patients with neuromyelitis optica
door: Nytrova, P, et al.
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Complement activation in patients with neuromyelitis optica.
door: Nytrova, P, et al.
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Plasmablasts as migratory IgG-producing cells in the pathogenesis of neuromyelitis optica.
door: Norio Chihara, et al.
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Differentiated pattern of complement system activation between MOG-IgG-associated disease and AQP4-IgG-positive neuromyelitis optica spectrum disorder
door: Eun Bin Cho, et al.
Gepubliceerd in: (2024-03-01)

Effect of IgG from seronegative and seropositive neuromyelitis optica patients on mouse brain
door: Saadoun, S, et al.
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Clinical fluctuations in MuSK myasthenia gravis are related to antigen-specific IgG4 instead of IgG1.
door: Niks, E, et al.
Gepubliceerd in: (2008)