Adhesion molecule interactions facilitate human immunodeficiency virus type 1-induced virological synapse formation between T cells.
Human immunodeficiency virus type 1 (HIV-1) can spread between CD4+ T cells by using a virological synapse (VS). The VS assembly is a cytoskeleton-driven process dependent on HIV-1 envelope glycoprotein (Env)-receptor engagement and is hypothesized to require adhesion molecule interactions. Here we...
Main Authors: | , , |
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Format: | Journal article |
Language: | English |
Published: |
2007
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