INDEPENDENT MECHANISMS FOR L-GLUTAMINE INFLUX IN NORMAL AND MALARIA-INFECTED HUMAN-ERYTHROCYTES

INDEPENDENT MECHANISMS FOR L-GLUTAMINE INFLUX IN NORMAL AND MALARIA-INFECTED HUMAN-ERYTHROCYTES

Bibliografiske detaljer
Main Authors:	Elford, B, Ellory, J, Newbold, C
Format:	Journal article
Udgivet:	1988

Lignende værker

Enhanced choline and Rb+ transport in human erythrocytes infected with the malaria parasite Plasmodium falciparum.
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Heterogeneous and substrate-specific membrane transport pathways induced in malaria-infected erythrocytes.
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A TRANSPORT PATHWAY RESPONSIBLE FOR THE INCREASED PERMEABILITY OF MALARIA-INFECTED ERYTHROCYTES SHOWS CHARACTERISTICS OF A CL- CHANNEL
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Transport of diverse substrates into malaria-infected erythrocytes via a pathway showing functional characteristics of a chloride channel.
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Udgivet: (1994)

Transport mechanisms across cell membranes.
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Udgivet: (1988)

TRANSPORT MECHANISMS ACROSS CELL-MEMBRANES - INTRODUCTORY-REMARKS
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Udgivet: (1988)

QUININE INHIBITS CATION-SELECTIVE MEMBRANE-TRANSPORT IN PLASMODIUM-FALCIPARUM-INFECTED HUMAN RED-CELLS
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Plasma glutamine levels and falciparum malaria.
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Characterization of the pathway for transport of the cytoadherence-mediating protein, PfEMP1, to the host cell surface in malaria parasite-infected erythrocytes.
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Identification of system y+L as the high-affinity transporter for L-arginine in human platelets: up-regulation of L-arginine influx in uraemia.
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INTERACTIONS OF MALARIA-INFECTED ERYTHROCYTES WITH ICAM-1 AND VASCULAR ENDOTHELIUM
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Surface changes to human erythrocytes on infection by Plasmodium falciparum malaria
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Calcium influx: An essential process by which α-Synuclein regulates morphology of erythrocytes
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Protection against clinical malaria by heterologous immunoglobulin G antibodies against malaria-infected erythrocyte variant surface antigens requires interaction with asymptomatic infections.
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Enhanced Ca2+ influx in mechanically distorted erythrocytes measured with 19F nuclear magnetic resonance spectroscopy
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Comediation of Erythrocyte Haemolysis by Erythrocyte-Derived Microparticles and Complement during Malaria Infection
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Functional analysis of molecular mechanisms associated with invasion of erythrocytes by human malaria parasite.
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Distribution of malaria parasite-derived phosphatidylcholine in the infected erythrocyte
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Metabolism of phospholipids in normal and spherocytic human erythrocytes
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Failure to respond to the surface of Plasmodium falciparum infected erythrocytes predicts susceptibility to clinical malaria amongst African children.
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Assessment of novel pre-erythrocytic malaria vaccines in humans
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Decreased free Mg2+ and pH in sickle erythrocytes
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Is the development of falciparum malaria in the human host limited by the availability of uninfected erythrocytes?
af: Hoshen M B, et al.
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Glutamine synthetase is a genetic determinant of cell type-specific glutamine independence in breast epithelia.
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Opsonization of malaria-infected erythrocytes activates the inflammasome and enhances inflammatory cytokine secretion by human macrophages
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Innate immune responses to malaria-infected erythrocytes in pregnant women: Effects of gravidity, malaria infection, and geographic location.
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High density of 'spiky' excrescences covering the surface of an erythrocyte infected with Plasmodium malariae.
af: Li, A, et al.
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Red cell selectivity in malaria: a study of multiple-infected erythrocytes.
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Targets of antibodies against Plasmodium falciparum-infected erythrocytes in malaria immunity.
af: Chan, J, et al.
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Structural studies of the PIR and RIFIN families from malaria-infected erythrocytes
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The effects of dyslipidaemia and cholesterol modulation on erythrocyte susceptibility to malaria parasite infection
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H-independent glutamine transport in plant root tips.
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Udgivet: (1990)

Human antibodies that slow erythrocyte invasion potentiate malaria-neutralizing antibodies
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