Identification of target mRNAs regulated by both p38 MAPK and tristetraprolin in murine macrophages

Identification of target mRNAs regulated by both p38 MAPK and tristetraprolin in murine macrophages

التفاصيل البيبلوغرافية
المؤلفون الرئيسيون:	Tudor, C, Hitti, E, Blackshear, P, Clark, A, Saklatvala, J, Dean, J
التنسيق:	Conference item
منشور في:	2007

مواد مشابهة

The p38 MAPK pathway inhibits tristetraprolin-directed decay of interleukin-10 and pro-inflammatory mediator mRNAs in murine macrophages.
حسب: Tudor, C, وآخرون
منشور في: (2009)

The stability of tristetraprolin mRNA is regulated by mitogen-activated protein kinase p38 and by tristetraprolin itself.
حسب: Tchen, C, وآخرون
منشور في: (2004)

The p38 MAPK pathway mediates both antiinflammatory and proinflammatory processes: comment on the article by Damjanov and the editorial by Genovese.
حسب: Clark, A, وآخرون
منشور في: (2009)

MAPKAP kinase 2 blocks tristetraprolin-directed mRNA decay by inhibiting CAF1 deadenylase recruitment.
حسب: Marchese, F, وآخرون
منشور في: (2010)

Mitogen-activated protein kinase p38 controls the expression and posttranslational modification of tristetraprolin, a regulator of tumor necrosis factor alpha mRNA stability.
حسب: Mahtani, K, وآخرون
منشور في: (2001)

p38 Mitogen-activated protein kinase stabilizes mRNAs that contain cyclooxygenase-2 and tumor necrosis factor AU-rich elements by inhibiting deadenylation.
حسب: Dean, J, وآخرون
منشور في: (2003)

PARP-14 combines with tristetraprolin in the selective posttranscriptional control of macrophage tissue factor expression.
حسب: Iqbal, M, وآخرون
منشور في: (2014)

Posttranslational regulation of tristetraprolin subcellular localization and protein stability by p38 mitogen-activated protein kinase and extracellular signal-regulated kinase pathways.
حسب: Brook, M, وآخرون
منشور في: (2006)

Palytoxin and an Ostreopsis toxin extract increase the levels of mRNAs encoding inflammation-related proteins in human macrophages via p38 MAPK and NF-κB.
حسب: Rita Crinelli, وآخرون
منشور في: (2012-01-01)

Identification of a major phosphopeptide in human tristetraprolin by phosphopeptide mapping and mass spectrometry.
حسب: Heping Cao, وآخرون
منشور في: (2014-01-01)

Differential upregulation of AU-rich element-containing mRNAs in COVID-19
حسب: Tala Bakheet, وآخرون
منشور في: (2022-11-01)

Dexamethasone causes sustained expression of mitogen-activated protein kinase (MAPK) phosphatase 1 and phosphatase-mediated inhibition of MAPK p38.
حسب: Lasa, M, وآخرون
منشور في: (2002)

Tristetraprolin mediates immune evasion of mycobacterial infection in macrophages
حسب: Jiawei Wei, وآخرون
منشور في: (2024-08-01)

Phosphorylation of human tristetraprolin in response to its interaction with the Cbl interacting protein CIN85.
حسب: Vishram P Kedar, وآخرون
منشور في: (2010-03-01)

The p38 MAPK Pathway in Rheumatoid Arthritis: A Sideways Look.
حسب: Clark, A, وآخرون
منشور في: (2012)

Tristetraprolin Prevents Gastric Metaplasia in Mice by Suppressing Pathogenic InflammationSummary
حسب: Jonathan T. Busada, وآخرون
منشور في: (2021-01-01)

MicroRNAs profiling in murine models of acute and chronic asthma: a relationship with mRNAs targets.
حسب: Nancy Garbacki, وآخرون
منشور في: (2011-01-01)

Global analysis of the abundance of AU-rich mRNAs in response to glucocorticoid treatment
حسب: Zeyad Muazzen, وآخرون
منشور في: (2024-01-01)

Tristetraprolin mediates radiation-induced TNF-α production in lung macrophages.
حسب: Dipankar Ray, وآخرون
منشور في: (2013-01-01)

Detection of mRNAs for macrophage products in inflammatory bowel disease by in situ hybridisation.
حسب: Cappello, M, وآخرون
منشور في: (1992)

Tristetraprolin expression by keratinocytes protects against skin carcinogenesis
حسب: Assiya Assabban, وآخرون
منشور في: (2021-03-01)

Tristetraprolin limits age-related expansion of myeloid-derived suppressor cells
حسب: Kyu Hwan Kwack, وآخرون
منشور في: (2022-10-01)

Post-transcriptional regulation of gene expression by mitogen-activated protein kinase p38.
حسب: Clark, A, وآخرون
منشور في: (2003)

Tristetraprolin Overexpression in Non-hematopoietic Cells Protects Against Acute Lung Injury in Mice
حسب: Ishita Choudhary, وآخرون
منشور في: (2020-09-01)

Lipopolysaccharide regulation of antiinflammatory tristetraprolin family and proinflammatory gene expression in mouse macrophages
حسب: Heping Cao
منشور في: (2024-03-01)

Tristetraprolin Regulates TH17 Cell Function and Ameliorates DSS-Induced Colitis in Mice
حسب: Hui Peng, وآخرون
منشور في: (2020-08-01)

Role of Tristetraprolin in the Resolution of Inflammation
حسب: Peter Rappl, وآخرون
منشور في: (2021-01-01)

Testosterone regulation of sex steroid-related mRNAs and dopamine-related mRNAs in adolescent male rat substantia nigra
حسب: Purves-Tyson Tertia D, وآخرون
منشور في: (2012-08-01)

To Be or Not to Be: Circular RNAs or mRNAs From Circular DNAs?
حسب: Leire Iparraguirre, وآخرون
منشور في: (2019-10-01)

Positive selection at codon 38 of the human <it>KCNE1 </it>(= <it>minK</it>) gene and sporadic absence of 38Ser-coding mRNAs in Gly38Ser heterozygotes
حسب: Pfeufer Arne, وآخرون
منشور في: (2009-08-01)

Multiple Export Mechanisms for mRNAs
حسب: Mildred Delaleau, وآخرون
منشور في: (2015-08-01)

Rapid quantification of murine ABC mRNAs by real time reverse transcriptase-polymerase chain reaction
حسب: Yan Ru Su, وآخرون
منشور في: (2002-12-01)

Tristetraprolin binding site atlas in the macrophage transcriptome reveals a switch for inflammation resolution
حسب: Vitaly Sedlyarov, وآخرون
منشور في: (2016-05-01)

Tristetraprolin inhibits macrophage IL-27-induced activation of antitumour cytotoxic T cell responses
حسب: Qinghong Wang, وآخرون
منشور في: (2017-10-01)

Translational regulation of specific mRNAs controls feedback inhibition and survival during macrophage activation.
حسب: Johanna Schott, وآخرون
منشور في: (2014-06-01)

Identification and analysis of long non-coding RNAs and mRNAs in chicken macrophages infected with avian infectious bronchitis coronavirus
حسب: Hao Li, وآخرون
منشور في: (2021-01-01)

Lack of STAT6 enhances murine acute lung injury through NLRP3/p38 MAPK signaling pathway in macrophages
حسب: Lu Hu, وآخرون
منشور في: (2022-05-01)

Gain-of-function mutation of tristetraprolin impairs negative feedback control of macrophages in vitro yet has overwhelmingly anti-inflammatory consequences in vivo
حسب: O'Neil, JD, وآخرون
منشور في: (2017)

Most mammalian mRNAs are conserved targets of microRNAs
حسب: Friedman, Robin Carl, وآخرون
منشور في: (2011)

Interplay of viral miRNAs and host mRNAs and proteins
حسب: Zhdanov Vladimir
منشور في: (2011-10-01)