Nitric oxide and H2O2 contribute to reactive dilation of isolated coronary arterioles.
The role of metabolic factors derived from cardiac muscle in the development of reactive hyperemia after brief occlusions of the coronary circulation seems to be well established. However, the contribution of occlusion-induced changes in hemodynamic forces to eliciting reactive hyperemia is less kno...
| Main Authors: | , |
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| Format: | Journal article |
| Language: | English |
| Published: |
2004
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