Shear stress insensitivity of endothelial nitric oxide synthase expression as a genetic risk factor for coronary heart disease.
Coronary heart disease (CHD) is based on the development of atherosclerosis in coronary arteries. Shear stress-induced endothelial nitric oxide (NO) release not only contributes to local blood pressure control but also effectively helps to retard atherosclerosis. Therefore, functionally relevant pol...
Main Authors: | Cattaruzza, M, Guzik, T, Słodowski, W, Pelvan, A, Becker, J, Halle, M, Buchwald, AB, Channon, K, Hecker, M |
---|---|
Format: | Journal article |
Language: | English |
Published: |
2004
|
Similar Items
-
Dihydrofolate reductase protects endothelial nitric oxide synthase from uncoupling in tetrahydrobiopterin deficiency
by: Crabtree, M, et al.
Published: (2011) -
Dihydrofolate reductase protects endothelial nitric oxide synthase from uncoupling in tetrahydrobiopterin deficiency.
by: Crabtree, M, et al.
Published: (2011) -
Tetrahydrobiopterin: regulator of endothelial nitric oxide synthase in vascular disease.
by: Channon, K
Published: (2004) -
Regulation of endothelial nitric oxide synthase by tetrahydrobiopterin in vascular disease.
by: Alp, N, et al.
Published: (2004) -
Relationship between the G894T polymorphism (Glu298Asp variant) in endothelial nitric oxide synthase and nitric oxide-mediated endothelial function in human atherosclerosis.
by: Guzik, T, et al.
Published: (2001)