Neuronal nitric oxide synthase (NOS1) regulates beta-adrenergic contraction and calcium handling in isolated murine ventricular myocytes

Neuronal nitric oxide synthase (NOS1) regulates beta-adrenergic contraction and calcium handling in isolated murine ventricular myocytes

מידע ביבליוגרפי
Main Authors:	Zhang, Y, Sears, C, Ashley, E, Kim, Y, Casadei, B
פורמט:	Conference item
יצא לאור:	2003

פריטים דומים

Cardiac nitric oxide synthase 1 regulates basal and beta-adrenergic contractility in murine ventricular myocytes.
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A cardiac neuronal nitric oxide synthase isoform regulates contraction and calcium handling in murine ventricular myocytes
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A cardiac neuronal nitric oxide synthase isoform regulates contraction and calcium handling in murine ventricular myocytes
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Enhancement of beta adrenergic responsiveness in myocytes lacking nitric oxide synthase 1
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Cardiac neuronal nitric oxide synthase isoform regulates myocardial contraction and calcium handling.
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Electrophysiological and mechanical properties of ventricular myocytes isolated from wild-type mice and mice with disruption of the neuronal nitric oxide synthase gene
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Does nitric oxide regulate the hyperpolarisation-activated inward current in ventricular myocytes from spontaneously hypertensive rats?
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Reduced phospholamban phosphorylation is associated with impaired relaxation in left ventricular myocytes from neuronal NO synthase-deficient mice.
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Effect of disruption of the nNOS gene on myocardial calcium current and contraction
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