GLUCOSE-INDUCED RETENTION OF MANGANESE IN PANCREATIC BETA-CELLS

GLUCOSE-INDUCED RETENTION OF MANGANESE IN PANCREATIC BETA-CELLS

التفاصيل البيبلوغرافية
المؤلف الرئيسي:	Rorsman, P
التنسيق:	Journal article
منشور في:	1982

مواد مشابهة

Glucose promotes intracellular sequestration of manganese in pancreatic beta-cells.
حسب: Rorsman, P
منشور في: (1982)

Manganese accumulation in pancreatic beta-cells and its stimulation by glucose.
حسب: Rorsman, P, وآخرون
منشور في: (1982)

The interaction between manganese and calcium fluxes in pancreatic beta-cells.
حسب: Rorsman, P, وآخرون
منشور في: (1983)

Glucose-sensing mechanisms in pancreatic beta-cells.
حسب: MacDonald, P, وآخرون
منشور في: (2005)

MANGANESE ACCUMULATION IN PANCREATIC B-CELLS DUE TO GLUCOSE INHIBITION OF OUTWARD TRANSPORT
حسب: Rorsman, P, وآخرون
منشور في: (1981)

EVIDENCE FOR A PRONOUNCED MITOCHONDRIAL ACCUMULATION OF MANGANESE IN GLUCOSE-STIMULATED PANCREATIC B-CELLS OF OB/OB MICE
حسب: Rorsman, P
منشور في: (1982)

EVIDENCE THAT GLUCOSE-INDUCED DEPOLARIZATION IN THE PANCREATIC BETA-CELLS INVOLVES THE CLOSURE OF AN ATP-DEPENDENT K+-CHANNEL
حسب: Rorsman, P, وآخرون
منشور في: (1985)

Glucose dependent K+-channels in pancreatic beta-cells are regulated by intracellular ATP.
حسب: Rorsman, P, وآخرون
منشور في: (1985)

Electrophysiology of the pancreatic beta-cell.
حسب: Ashcroft, F, وآخرون
منشور في: (1989)

Modulation of dihydropyridine-sensitive Ca2+ channels by glucose metabolism in mouse pancreatic beta-cells.
حسب: Smith, P, وآخرون
منشور في: (1989)

Dual effects of glucose on the cytosolic Ca2+ activity of mouse pancreatic beta-cells.
حسب: Rorsman, P, وآخرون
منشور في: (1984)

Glucose-dependent regulation of rhythmic action potential firing in pancreatic beta-cells by K(ATP)-channel modulation.
حسب: Kanno, T, وآخرون
منشور في: (2002)

Ca2+ transport in pancreatic beta-cells during glucose stimulation of insulin secretion.
حسب: Hellman, B, وآخرون
منشور في: (1980)

Insulin granule dynamics in pancreatic beta cells.
حسب: Rorsman, P, وآخرون
منشور في: (2003)

Direct evidence for opposite effects of D-glucose and D-glyceraldehyde on cytoplasmic pH of mouse pancreatic beta-cells.
حسب: Arkhammar, P, وآخرون
منشور في: (1986)

PVHL is a regulator of glucose metabolism and insulin secretion in pancreatic beta cells.
حسب: Zehetner, J, وآخرون
منشور في: (2008)

Simultaneous recordings of glucose dependent electrical activity and ATP-regulated K(+)-currents in isolated mouse pancreatic beta-cells.
حسب: Smith, P, وآخرون
منشور في: (1990)

Insulin secretion: Function and therapy of pancreatic beta-cells in diabetes
حسب: Rorsman, P
منشور في: (2005)

The pancreatic beta-cell as a fuel sensor: an electrophysiologist's viewpoint.
حسب: Rorsman, P
منشور في: (1997)

Glucose-induced increase in cytoplasmic pH in pancreatic beta-cells is mediated by Na+/H+ exchange, an effect not dependent on protein kinase C.
حسب: Juntti-Berggren, L, وآخرون
منشور في: (1991)

DIRECT EVIDENCE THAT GLUCOSE BOTH REDUCES AND INCREASES CYTOSOLIC FREE CA-2+ IN MOUSE PANCREATIC BETA-CELLS
حسب: Rorsman, P, وآخرون
منشور في: (1984)

Exocytotic properties of human pancreatic beta-cells.
حسب: Braun, M, وآخرون
منشور في: (2009)

Oscillations, intercellular coupling, and insulin secretion in pancreatic beta cells
حسب: Macdonald, P, وآخرون
منشور في: (2006)

Epigenetic Changes Induced by High Glucose in Human Pancreatic Beta Cells
حسب: Rasha A. Alhazzaa, وآخرون
منشور في: (2023-01-01)

Oscillations, intercellular coupling, and insulin secretion in pancreatic beta cells.
حسب: MacDonald, P, وآخرون
منشور في: (2006)

Stimulus-secretion coupling in pancreatic beta cells.
حسب: Ashcroft, F, وآخرون
منشور في: (1994)

The dual action of glucose on the cytosolic Ca2+ activity in pancreatic beta-cells. Demonstration of an inhibitory effect of glucose on insulin release in the mouse and man.
حسب: Hellman, B, وآخرون
منشور في: (1985)

ATP-dependence of exocytosis in mouse pancreatic beta-cells
حسب: Eliasson, L, وآخرون
منشور في: (1997)

SINGLE CALCIUM-CHANNEL ACTIVITY IN MOUSE PANCREATIC BETA-CELLS
حسب: Ashcroft, F, وآخرون
منشور في: (1989)

Single calcium channel activity in mouse pancreatic beta-cells.
حسب: Ashcroft, F, وآخرون
منشور في: (1989)

Failure of glucose to elicit a normal secretory response in fetal pancreatic beta cells results from glucose insensitivity of the ATP-regulated K+ channels.
حسب: Rorsman, P, وآخرون
منشور في: (1989)

DIRECT DETERMINATION OF MANGANESE IN MICROGRAM AMOUNTS OF PANCREATIC TISSUE BY ELECTROTHERMAL ATOMIC-ABSORPTION SPECTROMETRY
حسب: Rorsman, P, وآخرون
منشور في: (1982)

Regulated exocytosis of GABA from pancreatic beta-cells
حسب: Braun, M, وآخرون
منشور في: (2004)

Oscillations, intercellular coupling, and insulin secretion in pancreatic beta cells.
حسب: Patrick E MacDonald, وآخرون
منشور في: (2006-02-01)

Glucose induces closure of single potassium channels in isolated rat pancreatic beta-cells.
حسب: Ashcroft, F, وآخرون
منشور في: (1984)

Inositol trisphosphate-dependent periodic activation of a Ca(2+)-activated K+ conductance in glucose-stimulated pancreatic beta-cells.
حسب: Ammälä, C, وآخرون
منشور في: (1991)

THE ROLE OF ION CHANNELS IN STIMULUS-SECRETION COUPLING IN PANCREATIC BETA-CELLS
حسب: Ashcroft, F, وآخرون
منشور في: (1990)

Calcium and delayed potassium currents in mouse pancreatic beta-cells under voltage-clamp conditions.
حسب: Rorsman, P, وآخرون
منشور في: (1986)

Calcium and potassium currents recorded from pancreatic beta-cells under voltage clamp control.
حسب: Trube, G, وآخرون
منشور في: (1986)

The ins and outs of secretion from pancreatic beta-cells: control of single-vesicle exo- and endocytosis.
حسب: MacDonald, P, وآخرون
منشور في: (2007)