Tetrahydrobiopterin- and glutathionylation-dependent regulation of eNOS coupling: Relationships revealed by eNOS glutathionylation mutants in cells with tet-regulated GTP cyclohydrolase I expression
Prif Awduron: | Crabtree, M, Hale, A, Batchelor, H, Channon, K |
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Fformat: | Conference item |
Cyhoeddwyd: |
2012
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Eitemau Tebyg
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Quantitative regulation of intracellular endothelial nitric-oxide synthase (eNOS) coupling by both tetrahydrobiopterin-eNOS stoichiometry and biopterin redox status: insights from cells with tet-regulated GTP cyclohydrolase I expression.
gan: Crabtree, M, et al.
Cyhoeddwyd: (2009) -
Endothelial-targeted GTP cyclohydrolase I overexpression increases endothelial tetrahydrobiopterin levels and preserves eNOS coupling in eNOS overexpressing mice
gan: Bendall, J, et al.
Cyhoeddwyd: (2004) -
Tetrahydrobiopterin availability directly regulates eNOS coupling in tet-regulatable cells
gan: Crabtree, M, et al.
Cyhoeddwyd: (2007) -
Stoichiometric relationships between endothelial tetrahydrobiopterin, endothelial NO synthase (eNOS) activity, and eNOS coupling in vivo: insights from transgenic mice with endothelial-targeted GTP cyclohydrolase 1 and eNOS overexpression.
gan: Bendall, J, et al.
Cyhoeddwyd: (2005) -
Rapid Effects of Atorvastatin on Tetrahydrobiopterin-Mediated eNOS Coupling in Human Arteries, is Mediated by Stimulation of Vascular GTP-Cyclohydrolase
gan: Antoniades, C, et al.
Cyhoeddwyd: (2011)