Inhibition of lysosomal Ca2+ signalling disrupts dendritic spine structure and impairs wound healing in neurons
A growing body of evidence suggests that lysosomes, which have traditionally been regarded as degradative organelles, can function as Ca<sup>2+</sup> stores, regulated by the second messenger nicotinic acid adenine dinucleotide phosphate (NAADP). We previously demonstrated that in hippoc...
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| Format: | Journal article |
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Taylor and Francis Open
2017
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