The gata1/pu.1 lineage fate paradigm varies between blood populations and is modulated by tif1γ.

Lineage fate decisions underpin much of development as well as tissue homeostasis in the adult. A mechanistic paradigm for such decisions is the erythroid versus myeloid fate decision controlled by cross-antagonism between gata1 and pu.1 transcription factors. In this study, we have systematically t...

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Xehetasun bibliografikoak
Egile Nagusiak: Monteiro, R, Pouget, C, Patient, R
Formatua: Journal article
Hizkuntza:English
Argitaratua: 2011