Rapid emergence of chronic lymphocytic leukemia during JAK2 inhibitor therapy in a patient with myelofibrosis

Rapid emergence of chronic lymphocytic leukemia during JAK2 inhibitor therapy in a patient with myelofibrosis

Bibliográfalaš dieđut
Váldodahkkit:	Sousos, N, Buck, G, Rodriguez-Meira, A, Norfo, R, Hamblin, A, Pezzella, F, Davies, J, Hublitz, P, Psaila, B, Mead, AJ
Materiálatiipa:	Journal article
Giella:	English
Almmustuhtton:	Wolters Kluwer Health 2020

Geahča maid

Rapid Emergence of Chronic Lymphocytic Leukemia During JAK2 Inhibitor Therapy in a Patient With Myelofibrosis
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Correction Notice: Rapid Emergence of Chronic Lymphocytic Leukemia During JAK2 Inhibitor Therapy in a Patient With Myelofibrosis
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JAK inhibitors and myelofibrosis, Einstein and ruxolitinib
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Chronic Lymphocytic Leukemia and Myelofibrosis
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Treatment and management of myelofibrosis in the era of JAK inhibitors
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Almmustuhtton: (2013-08-01)

JAK Be Nimble: Reviewing the Development of JAK Inhibitors and JAK Inhibitor Combinations for Special Populations of Patients with Myelofibrosis
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Use of JAK inhibitors in the management of myelofibrosis: A revision of the British Committee for Standards in Haematology Guidelines for Investigation and Management of Myelofibrosis 2012
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Treatment and management of myelofibrosis in the era of JAK inhibitors [Corrigendum]
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Use of JAK inhibitors in the management of myelofibrosis: a revision of the British Committee for Standards in Haematology Guidelines for Investigation and Management of Myelofibrosis 2012.
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Is it primary myelofibrosis or chronic megakaryocytic leukemia?
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Risk‐adjusted safety analysis of the oral JAK2/IRAK1 inhibitor pacritinib in patients with myelofibrosis
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Comparative efficacy and hematologic safety of different dosages of JAK inhibitors in the treatment of myelofibrosis: a network meta-analysis
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Germline-somatic JAK2 interactions are associated with clonal expansion in myelofibrosis
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Acute massive myelofibrosis with acutelymphoblastic leukemia
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Primary Myelofibrosis Occurring during Targeted Therapy for Chronic Lymphocytic Leukemia: A Report of Two Cases
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A proinflammatory stem cell niche drives myelofibrosis through a targetable galectin-1 axis
Dahkki: Li, R, et al.
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A single-cell approach to unraveling abnormal megakaryocyte differentiation and function in myelofibrosis
Dahkki: Psaila, B, et al.
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Development of JAK2 positive myelofibrosis on a background of chronic myeloid leukaemia
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Frequency of JAK2 and MPL Mutation in BCR/ABL Negative Myelofibrosis in KPK
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The role of circulating monocytes and JAK inhibition in the infectious-driven inflammatory response of myelofibrosis
Dahkki: Martina Barone, et al.
Almmustuhtton: (2020-01-01)

Philadelphia chromosome positive AML arising from JAK2-positive myelofibrosis
Dahkki: Marte Karen Brattås, et al.
Almmustuhtton: (2018-11-01)

RAS-pathway mutations are common in patients with ruxolitinib refractory/intolerant myelofibrosis: molecular analysis of the PAC203 cohort
Dahkki: O'Sullivan, JM, et al.
Almmustuhtton: (2023)

CLINICAL AND PATHOMORPHOLOGICAL EXAMINATION OF MYELOFIBROSIS IN CHRONIC MYELOID LEUKEMIA
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Spliceosome mutations are common in persons with myeloproliferative neoplasm-associated myelofibrosis with RBC-transfusion-dependence and correlate with response to pomalidomide
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