Diabetes reduces vascular tetrahydrobiopterin levels by oxidation: Insights from a GTPCH-transgenic mouse model

Diabetes reduces vascular tetrahydrobiopterin levels by oxidation: Insights from a GTPCH-transgenic mouse model

Номзүйн дэлгэрэнгүй
Үндсэн зохиолчид:	Alp, N, Mussa, S, Goh, N, Cai, S, Channon, K
Формат:	Conference item
Хэвлэсэн:	2003

Ижил төстэй зүйлс

Increased endothelial tetrahydrobiopterin levels in GTPCH-transgenic mice
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Increased endothelial tetrahydrobiopterin levels by GTPCH over expression in transgenic mice prevents diabetic endothelial dysfunction
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Nitric oxide Bioavailability is preserved in diabetes by increased endothelial tetrahydrobiopterin synthesis in targeted GTP cyclohydrolase transgenic mice
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Tetrahydrobiopterin-dependent preservation of nitric oxide-mediated endothelial function in diabetes by targeted transgenic GTP-cyclohydrolase I overexpression.
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Regulation of endothelial nitric oxide synthase (eNOS) by tetrahydrobiopterin in vascular disease
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Regulation of endothelial nitric oxide synthase by tetrahydrobiopterin in vascular disease.
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Endothelial nitric oxide synthase dysfunction in diabetic mice: importance of tetrahydrobiopterin in eNOS dimerisation.
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Endothelial tetrahydrobiopterin regulates eNOS coupling/uncoupling independent of vascular disease: Insights from targeted transgenic models
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huGTPCH Expressing S plus S-Antilles Mice Have Reduced Gardos Channel Activity, Endothelin-1 Expression, and Endothelin Receptor A and B Expression, While Total Biopterin and Percent Tetrahydrobiopterin Are Increased
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The % tetrahydrobiopterin (BH4) and serine 1177 phosphorylation 19 reduced in sickle transgenic mice
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GTPCH I overexpression decreases atherosclerotic lesion formation in apolipoprotein E-deficient/eNOS transgenic mice
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Tetrahydrobiopterin: regulator of endothelial nitric oxide synthase in vascular disease.
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Tetrahydrobiopterin supplementation reduces atherosclerosis and vascular inflammation in apolipoprotein E-knockout mice.
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Congenic mapping and genotyping of the tetrahydrobiopterin-deficient hph-1 mouse.
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Tetrahydrobiopterin (BH4) Improves Endothelial Cell Survival After Vascular Injury
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Increased endothelial tetrahydrobiopterin synthesis by targeted transgenic GTP-cyclohydrolase I overexpression reduces endothelial dysfunction and atherosclerosis in ApoE-knockout mice.
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Increased endothelial tetrahydrobiopterin reduces in stent stenosis in apolipoprotein E-knockout mice
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Nitric oxide bioactivity in ApoE -/- mice is maintained by increased endothelial tetrahydrobiopterin synthesis via targeted GTP cyclohydrolase-1 over-expression
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Correction of tetrahydrobiopterin deficiency - Implications for dopa responsive dystonia and other inherited disorders of tetrahydrobiopterin metabolism
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The relative contributions of cardiomyocytes versus endothelial cells to cardiac BH4 production; studies in novel cardiomyocyte- and endothelial-specific GTPCH I-deficient mice
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Increased Endothelial Tetrahydrobiopterin Reduces In Stent Stenosis in Apolipoprotein E-Knockout Mice
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Regulation of eNOS function by tetrahydrobiopterin in vascular disease
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Prevention of hypoxia-induced pulmonary hypertension and vascular remodelling by targeting endothelial tetrahydrobiopterin-dependent nitric oxide synthase regulation
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Tetrahydrobiopterin and nitric oxide synthase recouplers
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Tetrahydrobiopterin deficiency increases susceptibility to atherosclerosis
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Synthesis and recycling of tetrahydrobiopterin in endothelial function and vascular disease.
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Synthesis and recycling of tetrahydrobiopterin in endothelial function and vascular disease
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Endothelial tetrahydrobiopterin deficiency accelerates atherosclerotic progression by nitric oxide synthase uncoupling
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Dihydrofolate reductase is required to maintain endothelial nitric oxide synthase coupling in vivo: Insights from methotrexate-treated BH4 deficient and GTPCH overexpressing mice
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Tetrahydrobiopterin: a vascular redox target to improve endothelial function.
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Low-dose Tetrahydrobiopterin Supplementation Reduces Atherosclerosis Progression in Apolipoprotein E-Knockout Mice via Effects on Vascular T cell Infiltration
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Increased endothelial tetrahydrobiopterin synthesis reduces vein graft atherosclerosis in ApoE-knockout mice
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Tetrahydrobiopterin deficiency uncouples nitric oxide synthase and causes pulmonary hypertension
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Stoichiometric relationships between endothelial tetrahydrobiopterin, endothelial NO synthase (eNOS) activity, and eNOS coupling in vivo: insights from transgenic mice with endothelial-targeted GTP cyclohydrolase 1 and eNOS overexpression.
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Tetrahydrobiopterin regulates vagal control of heart rate
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Tetrahydrobiopterin deficiency causes pulmonary hypertension: Effects on lung superoxide levels, hypoxic pulmonary vasoconstriction and vascular remodelling
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Tetrahydrobiopterin in the vascular system
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