Ketamine: Linking NMDA receptor hypofunction, gamma oscillations and psychosis (commentary on Qin et al., 2022)

Ketamine: Linking NMDA receptor hypofunction, gamma oscillations and psychosis (commentary on Qin et al., 2022)

Bibliografski detalji
Glavni autor:	Reilly, TJ
Format:	Journal article
Jezik:	English
Izdano:	Wiley 2022

Slični predmeti

NMDA receptor hypofunction leads to generalized and persistent aberrant gamma oscillations independent of hyperlocomotion and the state of consciousness.
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Effects of NMDA Receptor Hypofunction on Inhibitory Control in a Two-Layer Neural Circuit Model
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Fragile-X Syndrome Is Associated With NMDA Receptor Hypofunction and Reduced Dendritic Complexity in Mature Dentate Granule Cells
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Ketamine Infusion Associated with Improved Neurology in a Patient with NMDA Receptor Encephalitis
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Early Use of the NMDA Receptor Antagonist Ketamine in Refractory and Superrefractory Status Epilepticus
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Ketamine may block NMDA receptors in astrocytescausing a rapid antidepressant effect
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Parvalbumin expression and gamma oscillation occurrence increase over time in a neurodevelopmental model of NMDA receptor dysfunction
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Decision Making in Mice During an Optimized Touchscreen Spatial Working Memory Task Sensitive to Medial Prefrontal Cortex Inactivation and NMDA Receptor Hypofunction
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The Clinical Challenge of Autoimmune Psychosis: Learning from Anti-NMDA Receptor Autoantibodies
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Ketamine can produce oscillatory dynamics by engaging mechanisms dependent on the kinetics of NMDA receptors
od: Adam, Elie, i dr.
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od: Yu-Chin Hsiao, i dr.
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2-AG SIGNALING RECOVERS THE CB1 RECEPTOR-DEPENDENT LONG-TERM DEPRESSION OF THE PERFORANT PATH – GRANULE CELL SYNAPSE TRIGGERED BY NEONATAL HYPOFUNCTION OF NMDA RECEPTORS
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