Modification of superoxide-nitric oxide interactions in experimental venous bypass graft pathobiology by NOS gene transfer

Sonraí bibleagrafaíochta
Príomhchruthaitheoirí:	West, N, Guzik, T, Black, E, Channon, K
Formáid:	Journal article
Foilsithe / Cruthaithe:	2001

Nitric oxide synthase (nNOS) gene transfer modifies venous bypass graft remodeling: effects on vascular smooth muscle cell differentiation and superoxide production.
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Enhanced superoxide production in experimental venous bypass graft intimal hyperplasia: role of NAD(P)H oxidase.
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Enhanced superoxide anion production in experimental venous bypass grafts is mediated by NAD(P)H oxidase
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Superoxide generation by NAD(P)H oxidases in human bypass graft vessels.
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Superoxide production by NAD(P)H oxidases in human bypass graft vessels - Role in the regulation of vascular tone
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Endothelin-1 increases superoxide production in human coronary artery bypass grafts
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Nitric oxide produced by NOS2 copes with the cytotoxic effects of superoxide in macrophages
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Vascular superoxide generation by NAD(P)H oxidase is increased in humans with type II diabetes mellitus
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5-methyl-tetrahydrofolic acid increases nitric oxide bioavailability and decreases superoxide production by improving eNOS coupling in human vessels in vivo: Effects on intracellular tetrahydrobiopterin oxidation
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Chronic treatment with folic acid improves nitric oxide bioavailability and decreases superoxide production in human vessels, through tetrahydrobiopterin-mediated improvement of eNOS coupling
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Relation of preoperative radial artery flow-mediated dilatation to nitric oxide bioavailability in radial artery grafts used in off-pump coronary artery bypass grafting.
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5-methyl-tetrahydrofolate increases nitric oxide bioavailability and decreases superoxide production by improving eNOS coupling and preventing the intracellular oxidation of tetrahy-drobiopterin, in human vessels in vivo
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Genetic Polymorphism On Methyl-Tetrahydrofolate-Reductase Affects Vascular Superoxide Generation and Nitric Oxide Bioavailability By Regulating eNOS Coupling in Human Arteries and Veins
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The common genetic polymorphism C677T of methyltetrahydrofolate reductase regulates nitric oxide bioavailability and vascular superoxide production via modulation of eNOS coupling in human vessels
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Inhibition of superoxide and iNOS augment cutaneous nitric oxide‐dependent vasodilation in non‐Hispanic black young adults
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Functional impact of genetic polymorphisms in NAD(P)H oxidase p22phox subunit on vascular superoxide production in atherosclerosis
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Functional effect of the C242T polymorphism in the NAD(P)H oxidase p22phox gene on vascular superoxide production in atherosclerosis.
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Functional importance of C242T polymorphism in NAD(P)H oxidase p22phox subunit on vascular superoxide production in atherosclerosis
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Plasma asymmetrical-dimethyl-arginine (ADMA) regulates nitric oxide bioavailability and vascular superoxide production in human vessels
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Preoperative evaluation of flow-mediated dilatation in the left radial artery or the right brachial artery, as a predictor of nitric-oxide bioavailability in coronary artery bypass grafts
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Modification of superoxide-nitric oxide interactions in experimental venous bypass graft pathobiology by NOS gene transfer

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