GTP cyclohydrolase I/BH4 pathway protects EPCs via suppressing oxidative stress and thrombospondin-1 in salt-sensitive hypertension.

Endothelial progenitor cells (EPCs) are both reduced and dysfunctional in hypertension that correlates inversely with its mortality, but the mechanisms are poorly understood. Endothelial nitric oxide synthase (eNOS) critically regulates EPC mobilization and function but is uncoupled in salt-sensitiv...

Täydet tiedot

Bibliografiset tiedot
Päätekijät:	Xie, H, Zhou, S, Chen, D, Channon, K, Su, D, Chen, A
Aineistotyyppi:	Journal article
Kieli:	English
Julkaistu:	2010

GTP cyclohydrolase I/BH4 pathway protects EPCs via suppressing oxidative stress and thrombospondin-1 in salt-sensitive hypertension Tekijä Xie, H, Zhou, S, Chen, D, Channon, K, Su, D, Chen, A

Julkaistu 2010

Journal article

GTP cyclohydrolase I/BH4 pathway protects EPCs via suppressing oxidative stress and thrombospondin-1 in salt-sensitive hypertension.

GTP cyclohydrolase I/BH4 pathway protects EPCs via suppressing oxidative stress and thrombospondin-1 in salt-sensitive hypertension Tekijä Xie, H, Zhou, S, Chen, D, Channon, K, Su, D, Chen, A

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