Chondroclasts are mature osteoclasts which are capable of cartilage matrix resorption.

Chondroclasts are mature osteoclasts which are capable of cartilage matrix resorption.

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Multinucleated cells termed chondroclasts have been observed on the deep surface of resorbed hyaline cartilage but the relationship of these cells to macrophages and osteoclasts and their role in rheumatoid arthritis (RA) and other arthritic conditions is uncertain. Multinucleated cells in RA and ot...

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Príomhchruthaitheoirí:	Knowles, H, Moskovsky, L, Thompson, M, Grunhen, J, Cheng, X, Kashima, T, Athanasou, N
Formáid:	Conference item
Foilsithe / Cruthaithe:	2012

Míreanna comhchosúla

Chondroclasts are mature osteoclasts which are capable of cartilage matrix resorption
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Foilsithe / Cruthaithe: (2012)

Synovial fluid macrophages are capable of osteoclast formation and resorption.
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Acute hypoxia and osteoclast activity: a balance between enhanced resorption and increased apoptosis.
de réir: Knowles, H, et al.
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The Role of Calcium and Nicotinic Acid Adenine Dinucleotide Phosphate (NAADP) in Human Osteoclast Formation and Resorption.
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In vitro generation of mature human osteoclasts.
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Effects of acute hypoxia on osteoclast activity: A balance between enhanced resorption and increased apoptosis
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Human osteoclast ontogeny and pathological bone resorption.
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Effects of hypoxia on osteoclast formation and function: A balance between enhanced resorptive activity and increased apoptosis
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Recent advances in osteoclast biology and pathological bone resorption.
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Osteoclast differentiation and bone resorption in multicentric reticulohistiocytosis.
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Hypoxia-inducible factor regulates osteoclast-mediated bone resorption: role of angiopoietin-like 4.
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Hypoxic regulation of osteoclast differentiation and bone resorption activity
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Two distinct cellular mechanisms of osteoclast formation and bone resorption in periprosthetic osteolysis.
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Osteoclasts degrade bone and cartilage knee joint compartments through different resorption processes
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Osteoclast formation and function in pigmented villonodular synovitis
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Osteoclast precursors at sites of pathological bone resorption are CD14+mononuclear phagocytes
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Cellular and humoral mechanisms of osteoclast formation and bone resorption in Gorham-Stout disease.
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Rankl-independent induction of human osteoclast formation and bone resorption by TGF-beta
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Hypoxia-induced fibroblast growth factor 11 stimulates osteoclast-mediated resorption of bone
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