Five quantitative trait loci control radiation-induced adenoma multiplicity in Mom 1R ApcMin/+ mice
Ionising radiation is a carcinogen capable of inducing tumours, including colorectal cancer, in both humans and animals. By backcrossing a recombinant line of ApcMin/+ mice to the inbred BALB/c mouse strain, which is unusually sensitive to radiation-induced tumour development, we obtained panels of...
المؤلفون الرئيسيون: | Elahi, E, Suraweera, N, Volikos, E, Haines, J, Brown, N, Davidson, G, Churchman, M, Ilyas, M, Tomlinson, I, Silver, A |
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التنسيق: | Journal article |
اللغة: | English |
منشور في: |
Public Library of Science
2009
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مواد مشابهة
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Five quantitative trait loci control radiation-induced adenoma multiplicity in Mom1R Apc Min/+ mice.
حسب: Elahi, E, وآخرون
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Five quantitative trait loci control radiation-induced adenoma multiplicity in Mom1R Apc Min/+ mice.
حسب: Eiram Elahi, وآخرون
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Genetic basis of variation in adenoma multiplicity in ApcMin/+ Mom1S mice.
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Myh deficiency enhances intestinal tumorigenesis in multiple intestinal neoplasia (ApcMin/+) mice.
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Platelet status in cancer cachexia progression in ApcMin/+ mice
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منشور في: (2023-08-01)