The mechanism of alpha-globin activation during erythropoiesis

The mechanism of alpha-globin activation during erythropoiesis

Podrobná bibliografie
Hlavní autoři:	Higgs, D, Vernimmen, D, Ayyub, H, De Gobbi, M, Hughes, J, Dunham, I, Koch, C, Buckle, V, Iborra, F, Brown, J, Sharpe, J, Sloane-Stanley, J, Gibbons, R, Wood, B
Médium:	Conference item
Vydáno:	2007

Podobné jednotky

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Deletion of the mouse alpha-globin regulatory element (HS -26) has an unexpectedly mild phenotype.
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The mouse alpha-globin locus regulatory element.
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Analysis of the human alpha globin upstream regulatory element (HS-40) in transgenic mice.
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Understanding alpha-globin gene regulation: Aiming to improve the management of thalassemia.
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A large deletion in the human alpha-globin cluster caused by a replication error is associated with an unexpectedly mild phenotype.
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Spatial and temporal organisation of globin genes during erytroid differentiation.
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A 16.5 kb deletion in the alpha globin cluster associated with an extremely mild phenotype
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Analysis of the human alpha-globin gene cluster in transgenic mice.
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Chapter 5 Long-Range Regulation of α-Globin Gene Expression
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Dynamic changes in transcription factor binding and epigenetic modification during erythropoiesis.
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Molecular rearrangements of the human alpha-globin gene cluster.
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Coregulated human globin genes are frequently in spatial proximity when active
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GEOGRAPHIC-DISTRIBUTION OF ALPHA-GLOBIN AND BETA-GLOBIN GENE-CLUSTER POLYMORPHISMS
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A METHOD FOR THE HIGH-RESOLUTION IDENTIFICATION OF ACTIVE CIS ELEMENTS FOR ANY GIVEN GENE AND ITS APPLICATION TO THE HUMAN ALPHA GLOBIN LOCUS
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Role of upstream DNase I hypersensitive sites in the regulation of human alpha globin gene expression.
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Analysis of sequence variation underlying tissue-specific transcription factor binding and gene expression.
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Deletion of the alpha-globin gene cluster as a cause of acquired alpha-thalassemia in myelodysplastic syndrome.
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Long-range chromosomal interactions regulate the timing of the transition between poised and active gene expression
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Long-range chromosomal interactions regulate the timing of the transition between poised and active gene expression.
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Alpha globin genotypes in two North European populations.
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Codanin-1 Mutations In Congenital Dyserythropoietic Anemia Type 1 Affect HP1 alpha Localization In Erythroblasts
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