Visualising the dynamics of single insulin-containing granules undergoing exoctosis

Visualising the dynamics of single insulin-containing granules undergoing exoctosis

Bibliográfalaš dieđut
Váldodahkkit:	Barg, S, Olofsson, C, Schriever-Abeln, J, Wendt, A, Renstrom, E, Rorsman, P, Gebre-Medhin, S
Materiálatiipa:	Journal article
Almmustuhtton:	2001

Geahča maid

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Insulin granule dynamics in pancreatic beta cells.
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Regulation of insulin granule exocytosis
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Priming of insulin granules for exocytosis by granular Cl(-) uptake and acidification.
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A subset of 50 secretory granules in close contact with L-type Ca2+ channels accounts for first-phase insulin secretion in mouse beta-cells.
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Fast insulin secretion reflects exocytosis of docked granules in mouse pancreatic B-cells.
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Cyclic AMP stimulates insulin exocytosis by modification of granules already present at the release sites
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Fast exocytosis by Ca2+-channel-granule interaction in mouse pancreatic B-cells
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Ca2+ channel clustering with insulin-containing granules is disturbed in type 2 diabetes
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Physical interaction between secretory granules and L-type calcium channels is required for fast exocytosis in B-cells
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Selective nucleotide-release from dense-core granules in insulin-secreting cells.
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Role for [Ca2+] microdomains in glucose-induced movement of GFP-tagged secretory granules.
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The stimulatory action of tolbutamide on Ca2+-dependent exocytosis is mediated by a 65-kDa mdr-like P-glycoprotein and ClC3Cl(-)-channels in granule membranes of insulin secreting B-cells.
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Diabetes - when insulin is trapped in the secretory granule?
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Sulfonylurea-mediated stimulation of insulin exocytosis via an ATP-sensitive K+ channel-independent action.
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The Cell Physiology of Biphasic Insulin Secretion.
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Regulated exocytosis of single insulin granules and synaptic-like vesicles in membrane patches of insulin secreting cells
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Exocytosis depends on granular chloride fluxes regulated by a sulfonylurea receptor-like protein in insulin-secreting B-cells
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Chromogranin-B deficient mice are diabetic despite normal insulin granule biosynthesis
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Cooling inhibits exocytosis in single mouse pancreatic B-cells by suppression of granule mobilization.
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Defective insulin release machinery and insulin granule acidification in chloride channel 3null (ClC3(-/-)) mice
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COOLING INHIBITS INSULIN-SECRETION BY SUPPRESSION OF GRANULE MOBILIZATION
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Fast exocytosis with few Ca2+-channels by economical use of Ca2+ in insulin-secreting pancreatic B-cells
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CAPS1 and CAPS2 regulate stability and recruitment of insulin granules in mouse pancreatic beta cells.
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Insulin secretion: a high-affinity Ca2+ sensor after all?
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Regulated insulin release requires chloride channel 3 (ClC3) for proper granule acidification and priming
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Adrenaline stimulates glucagon secretion in pancreatic A-cells by increasing the Ca2+ current and the number of granules close to the L-type Ca2+ channels.
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Intracellularly applied ion channel modulators interfere with calcium induced exocytosis in mouse pancreatic B-cells
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Long-term exposure to glucose and lipids inhibits glucose-induced insulin secretion downstream of granule fusion with plasma membrane.
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Alternative MDR splicing is required for direct potentiation of exocytosis by sulphonylureas
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SUR1 regulates PKA-independent cAMP-induced granule priming in mouse pancreatic B-cells.
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Components of insulin secretion: lessons to be learnt from capacitance.
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Palmitate increases L-type Ca2+ currents and the size of the readily releasable granule pool in mouse pancreatic beta-cells.
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Inside the Insulin Secretory Granule
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Somatostatin but not insulin is an important paracrine regulator of glucagon secretion in rat A-cells
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Single vesicle measurement reveals Ca++ and cAMP dependent exocytosis of GABA from insulin-producing B-cells
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Identification and characterization of single glucagon-secreting A-cells in a standard mouse islet preparation
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