Skip to content
VuFind
English
Deutsch
Español
Français
Italiano
日本語
Nederlands
Português
Português (Brasil)
中文(简体)
中文(繁體)
Türkçe
עברית
Gaeilge
Cymraeg
Ελληνικά
Català
Euskara
Русский
Čeština
Suomi
Svenska
polski
Dansk
slovenščina
اللغة العربية
বাংলা
Galego
Tiếng Việt
Hrvatski
हिंदी
Հայերէն
Українська
Sámegiella
Монгол
Language
All Fields
Title
Author
Subject
Call Number
ISBN/ISSN
Tag
Find
Advanced
Mutations in ATP-sensitive K+...
Cite this
Text this
Email this
Print
Export Record
Export to RefWorks
Export to EndNoteWeb
Export to EndNote
Permanent link
Mutations in ATP-sensitive K+ channel genes cause transient neonatal diabetes and permanent diabetes in childhood or adulthood (vol 56, pg 1930, 2007)
Bibliographic Details
Main Authors:
Flanagan, SE
,
Patch, A
,
Mackay, D
,
Edghill, E
,
Gloyn, A
,
Robinson, D
,
Shield, J
,
Temple, K
,
Ellard, S
,
Hattersley, A
Format:
Journal article
Published:
2008
Holdings
Description
Similar Items
Staff View
Similar Items
Erratum: Mutations in ATP-sensitive K+ cannel genes cause transient neonatal diabetes and permanent diabetes in childhood or adulthood (Diabetes (2007) 56 (1930-1937))
by: Flanagan, SE, et al.
Published: (2008)
Mutations in ATP-sensitive K+ channel genes cause transient neonatal diabetes and permanent diabetes in childhood or adulthood.
by: Flanagan, SE, et al.
Published: (2007)
Complete glucokinase deficiency is not a common cause of permanent neonatal diabetes.
by: Gloyn, A, et al.
Published: (2002)
Mutations in KCNJ11, which encodes Kir6.2, are a common cause of diabetes diagnosed in the first 6 months of life, with the phenotype determined by genotype.
by: Flanagan, SE, et al.
Published: (2006)
Permanent neonatal diabetes caused by dominant, recessive, or compound heterozygous SUR1 mutations with opposite functional effects.
by: Ellard, S, et al.
Published: (2007)