Multiple Interactions Recruit MLL1 and MLL1 Fusion Proteins to the HOXA9 Locus in Leukemogenesis

Multiple Interactions Recruit MLL1 and MLL1 Fusion Proteins to the HOXA9 Locus in Leukemogenesis

عرض إصدارات أخرى (1)

التفاصيل البيبلوغرافية
المؤلفون الرئيسيون:	Milne, T, Kim, J, Wang, G, Stadler, S, Basrur, V, Whitcomb, S, Wang, Z, Ruthenburg, A, Elenitoba-Johnson, K, Roeder, R, Allis, C
التنسيق:	Journal article
منشور في:	2010

مواد مشابهة

Multiple interactions recruit MLL1 and MLL1 fusion proteins to the HOXA9 locus in leukemogenesis.
حسب: Milne, T, وآخرون
منشور في: (2010)

Function of leukemogenic mixed lineage leukemia 1 (MLL) fusion proteins through distinct partner protein complexes.
حسب: Biswas, D, وآخرون
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Molecular and Epigenetic Mechanisms of MLL in Human Leukemogenesis.
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PAF1 and FACT cooperate with MLL-AF4 to drive enhancer activity in leukemia
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Regulation of MLL1 H3K4 methyltransferase activity by its core components.
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منشور في: (2006)

The Histone Methyltransferase Activity of MLL1 Is Dispensable for Hematopoiesis and Leukemogenesis
حسب: Bibhu P. Mishra, وآخرون
منشور في: (2014-05-01)

Hoxa9 and Meis1 are key targets for MLL-ENL-mediated cellular immortalization.
حسب: Zeisig, B, وآخرون
منشور في: (2004)

Extending the repertoire of the mixed-lineage leukemia gene MLL in leukemogenesis.
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MLL-AF4 cooperates with PAF1 and FACT to drive high-density enhancer interactions in leukemia
حسب: Nicholas T. Crump, وآخرون
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Loss of MBD2 attenuates MLL-AF9-driven leukemogenesis by suppressing the leukemic cell cycle via CDKN1C
حسب: Kuangguo Zhou, وآخرون
منشور في: (2021-11-01)

Development of a multi-step leukemogenesis model of MLL-rearranged leukemia using humanized mice.
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An Ash2L/RbBP5 heterodimer stimulates the MLL1 methyltransferase activity through coordinated substrate interactions with the MLL1 SET domain.
حسب: Fang Cao, وآخرون
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TIFAB accelerates MLL-AF9−Induced acute myeloid leukemia through upregulation of HOXA9
حسب: Jinming Zhao, وآخرون
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Systematic characterization of the HOXA9 downstream targets in MLL-r leukemia by noncoding CRISPR screens
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Dimerization of MLL fusion proteins immortalizes hematopoietic cells.
حسب: Martin, M, وآخرون
منشور في: (2003)

Cdx4 is dispensable for murine adult hematopoietic stem cells but promotes MLL-AF9-mediated leukemogenesis
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Identification of genes transcriptionally responsive to the loss of MLL fusions in MLL-rearranged acute lymphoblastic leukemia.
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منشور في: (2015-01-01)

Essential Functions of MLL1 and MLL2 in Retinal Development and Cone Cell Maintenance
حسب: Chi Sun, وآخرون
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MLL targets SET domain methyltransferase activity to Hox gene promoters.
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The MLL family of proteins in normal development and disease
حسب: Milne, TA
منشور في: (2020)

MLL associates specifically with a subset of transcriptionally active target genes.
حسب: Milne, T, وآخرون
منشور في: (2005)

Genome-wide analyses reveal new insights into MLL fusion protein recruitment mechanisms
حسب: Kerry, J
منشور في: (2015)

Registered report: Inhibition of BET recruitment to chromatin as an effective treatment for MLL-fusion leukemia
حسب: Juan José Fung, وآخرون
منشور في: (2015-09-01)

Replication Study: Inhibition of BET recruitment to chromatin as an effective treatment for MLL-fusion leukaemia
حسب: Xiaochuan Shan, وآخرون
منشور في: (2017-06-01)

Research progress in Menin-MLL interaction and its inhibitors in MLL-rearranged leukemia
حسب: FANG Xinyue, وآخرون
منشور في: (2024-10-01)

Histone H3K36me2-Specific Methyltransferase ASH1L Promotes MLL-AF9-Induced Leukemogenesis
حسب: Mohammad B. Aljazi, وآخرون
منشور في: (2021-10-01)

Molecular characterization of an MLL1 fusion and its role in chromosomal instability
حسب: Sreejit Parameswaran, وآخرون
منشور في: (2019-02-01)

Physical association and coordinate function of the H3 K4 methyltransferase MLL1 and the H4 K16 acetyltransferase MOF.
حسب: Dou, Y, وآخرون
منشور في: (2005)

Leukemogenic MLL fusion proteins bind across a broad region of the Hox a9 locus, promoting transcription and multiple histone modifications.
حسب: Milne, T, وآخرون
منشور في: (2005)

Correction: Replication Study: Inhibition of BET recruitment to chromatin as an effective treatment for MLL-fusion leukaemia
حسب: Xiaochuan Shan, وآخرون
منشور في: (2018-01-01)

MLL3 regulates the CDKN2A tumor suppressor locus in liver cancer
حسب: Changyu Zhu, وآخرون
منشور في: (2023-06-01)

Trithorax and polycomb cooperation in MLL fusion acute leukemia
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FOXQ1 recruits the MLL complex to activate transcription of EMT and promote breast cancer metastasis
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RUNX1 is a key target in t(4;11) leukemias that contributes to gene activation through an AF4-MLL complex interaction.
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منشور في: (2013)

Unlike its Paralog LEDGF/p75, HRP-2 Is Dispensable for MLL-R Leukemogenesis but Important for Leukemic Cell Survival
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The deficiency of tumor suppressor prep1 accelerates the onset of meis1- hoxa9 leukemogenesis.
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MLL rearrangements in pediatric acute lymphoblastic and myeloblastic leukemias: MLL specific and lineage specific signatures
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