Consequences of presynaptic dysfunction in Alzheimer’s Disease models
<p>Soluble oligomeric assemblies of amyloid β (Aβo) drive much of Alzheimer’s disease (AD) pathology. Among these, changes in synaptic plasticity including attenuated long-term potentiation (LTP), and facilitated long-term depression (LTD), accompanied by postsynaptic weakening, are believed t...
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| Định dạng: | Luận văn |
| Ngôn ngữ: | English |
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2020
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