Consequences of presynaptic dysfunction in Alzheimer’s Disease models
<p>Soluble oligomeric assemblies of amyloid β (Aβo) drive much of Alzheimer’s disease (AD) pathology. Among these, changes in synaptic plasticity including attenuated long-term potentiation (LTP), and facilitated long-term depression (LTD), accompanied by postsynaptic weakening, are believed t...
| Main Author: | |
|---|---|
| Other Authors: | |
| Format: | Thesis |
| Language: | English |
| Published: |
2020
|
| Subjects: |