Transient elevation of glucose concentration reduces NO-mediation of flow-induced arteriolar dilation due to interaction between superoxide and tetrahydrobiopterin (BH4)

Παρόμοια τεκμήρια

• Impaired NO-mediation of flow dependent arteriolar dilation in diabetes due to reduced level of tetrahydrobiopterin
  ανά: Bagi, Z, κ.ά.
  Έκδοση: (2002)
• Microvascular dysfunction after transient high glucose is caused by superoxide-dependent reduction in the bioavailability of NO and BH(4).
  ανά: Bagi, Z, κ.ά.
  Έκδοση: (2004)
• Tetrahydrobiopterin (BH4) is essential for NO-mediated flow-dependent dilation of isolated skeletal muscle arterioles
  ανά: Bagi, Z, κ.ά.
  Έκδοση: (2003)
• Obesity-induced reduction in nitric oxide-dependent arteriolar dilation in rats. Role of xanthine oxidase-derived superoxide.
  ανά: Erdei, N, κ.ά.
  Έκδοση: (2006)
• Asymmetrical dimethylarginine inhibits shear stress-induced nitric oxide release and dilation and elicits superoxide-mediated increase in arteriolar tone.
  ανά: Toth, J, κ.ά.
  Έκδοση: (2007)