Cardiomyopathy-causing mutations in cardiac thin filament regulatory proteins acutely affect Ca2+ buffering and Ca2+ dependant signalling in situ

Míreanna comhchosúla

• Cardiomyopathy-Causing Mutations in Thin Filament Regulatory Proteins Acutely Affeect Ca2+-Buffering and Ca2+-Dependent Signalling In Situ
  de réir: Robinson, P, et al.
  Foilsithe / Cruthaithe: (2013)
• Investigating the Effect of Cardiomyopathy-Causing Mutations in Cardiac Troponin-T on Calcium Buffering In Situ
  de réir: Robinson, P, et al.
  Foilsithe / Cruthaithe: (2010)
• Hypertrophic cardiomyopathy mutations increase myofilament Ca2+ buffering, alter intracellular Ca2+ handling, and stimulate Ca2+-dependent signaling
  de réir: Robinson, P, et al.
  Foilsithe / Cruthaithe: (2018)
• Mavacamten rescues increased myofilament calcium sensitivity and dysregulation of Ca2+ flux caused by thin filament hypertrophic cardiomyopathy mutations
  de réir: Sparrow, AJ, et al.
  Foilsithe / Cruthaithe: (2020)
• Using fluorescence emission spectroscopy to asses the Ca2+ affinity of reconstituted troponin and thin filaments containing cardiomyopathy causing mutations of troponin and alpha-tropomyosin
  de réir: Robinson, P, et al.
  Foilsithe / Cruthaithe: (2007)