Atorvastatin rapidly modifies vascular redox state and improves nitric oxide bioavailability in human vessels obtained from patients with advanced atherosclerosis

מידע ביבליוגרפי
Main Authors:	Demosthenous, M, Antoniades, C, Paschalis, A, Tousoulis, D, Bakogiannis, C, Antonopoulos, A, Lymperiadis, D, Paleopoulos, T, Channon, K, Stefanadis, C
פורמט:	Conference item
יצא לאור:	2010

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Atorvastatin directly improves vascular redox state by affecting eNOS coupling and NADPH-oxidase activity in human vein grafts ex-vivo
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Short-Term Atorvastatin Treatment Reduces Vascular Oxidative Stress Saphenous Vein Grafts by Improving eNOS Coupling and Reducing NADPH-Oxidase Activity in Patients Undergoing Elective CABG
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Plasma asymmetrical-dimethyl-arginine (ADMA) regulates nitric oxide bioavailability and vascular superoxide production in human vessels
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Preoperative atorvastatin treatment in CABG patients rapidly improves vein graft redox state by inhibition of Rac1 and NADPH-oxidase activity.
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Short-term preoperative treatment with atorvastatin improves myocardial redox state in patients undergoing elective CABG by affecting NADPH oxidase activity and NOS coupling
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Polymorphism of MTHFR gene affects vascular redox and nitric oxide bioavailability by modifying eNOS coupling
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NADPH-oxidase activity in peripheral blood mononuclear cells as a novel inflammatory marker in atherosclerosis
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Atorvastatin rapidly reduces myocardial superoxide and peroxynitrite generation in human atherosclerosis, by suppressing NADPH-oxidase activity independently of LDL lowering
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Plasma asymmetrical-dimethyl-arginine affects eNOS vascular superoxide radical production and nitric oxide bioavailability in human arteries and veins
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Myocardial Redox State Predicts Post-Operative Atrial Fibrillation in Cardiac Surgery
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Homocysteine and coronary atherosclerosis: from folate fortification to the recent clinical trials.
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Atorvastatin rapidly modifies vascular redox state and improves nitric oxide bioavailability in human vessels obtained from patients with advanced atherosclerosis

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