NAADP influences excitation-contraction coupling by releasing calcium from lysosomes in atrial myocytes

NAADP influences excitation-contraction coupling by releasing calcium from lysosomes in atrial myocytes

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In atrial myocytes, the sarcoplasmic reticulum (SR) has an essential role in regulating the force of contraction as a consequence of its involvement in excitation-contraction coupling (ECC). Nicotinic acid adenine dinucleotide phosphate (NAADP) is a Ca 2+ mobilizing messenge...

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Xehetasun bibliografikoak
Egile Nagusiak:	Collins, T, Bayliss, R, Churchill, G, Galione, A, Terrar, D
Formatua:	Journal article
Hizkuntza:	English
Argitaratua:	2011

Antzeko izenburuak

NAADP influences excitation-contraction coupling by releasing calcium from lysosomes in atrial myocytes.
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NAADP influences excitation-contraction coupling by releasing calcium from lysosomes in atrial myocytes
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NAADP is a Physiologically-Relevant Calcium Mobilising Compound in Atrial Myocytes
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Actions of a membrane permeant analogue of NAADP on contractions and calcium sparks in cardiac ventricular myocytes.
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NAADP as an intracellular messenger regulating lysosomal calcium-release channels.
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NAADP-induced calcium release in sea urchin eggs.
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Actions of NAADP and other agents in cardiac myocytes
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NAADP: a novel Ca2+ mobilizing messenger during beta-adrenoceptor stimulation in guinea-pig ventricular myocytes
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Calcium signalling by nicotinic acid adenine dinucleotide phosphate (NAADP).
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Synthesis, degradation, binding and actions of NAADP in cardiac tissue.
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Effect of cyclic ADP-ribose on calcium transients and contractions in guinea-pig ventricular myocytes without an increase in the loading of the sarcoplasmic reticulum with calcium
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Actions of cADP-ribose phosphate on contraction in guinea-pig isolated ventricular myocytes
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Biphasic actions of cADP-ribose phosphate on contraction in guinea-pig isolated ventricular myocytes
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The influence of nicotinic acid adenine dinucleotide phosphate (NAADP) signalling and the significance of the organization of lysosomes in cardiac myocytes
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Coordination of Ca2+ signalling by NAADP.
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Unique kinetics of nicotinic acid-adenine dinucleotide phosphate (NAADP) binding enhance the sensitivity of NAADP receptors for their ligand.
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NAADP mobilizes Ca(2+) from reserve granules, lysosome-related organelles, in sea urchin eggs.
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Calcium stimulates the synthesis of nicotinic acid adenine dinucleotide phosphate (NAADP) in sea urchin sperm
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The Involvement of NAADP and Two-Pore Ca2+ Channels in the Cardiac Beta-Adrenergic Response
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Sperm expresses a surface bound base exchange enzyme that selectively synthesises NAADP and is controlled by calcium.
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NAADP receptors.
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NAADP Receptors
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NAADP receptors
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NAADP controls cross-talk between distinct Ca2+ stores in the heart.
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Regulation and pathway of NAADP synthesis in sea urchin sperm
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NAADP receptors.
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Cell-permeant small-molecule modulators of NAADP-mediated Ca2+ release.
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Sperm express a Ca2+-regulated NAADP synthase.
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Sperm expresses surface bound base exchange enzyme that selectively synthesises NAADP
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Rapid, selective and dose-dependent elevation of the second messenger NAADP
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Essential role of NAADP-evoked calcium release in glucose-mediated depolarization, [Ca2+]i spiking and insulin secretion in mouse pancreatic beta cell
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NAADP-regulated two-pore channels drive phagocytosis through endo-lysosomal Ca2+ nanodomains, calcineurin and dynamin
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A novel signalling role for NAADP in arterial smooth muscle
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Does NAADP-induced Ca2+ release contribute to glucose-induced (ca(2+))(i) changes in primary mouse pancreatic beta-cell?
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Intracellular sphingosine releases calcium from lysosomes
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Ca(2+)-stimulated adenylyl cyclases regulate the L-type Ca(2+) current in guinea-pig atrial myocytes.
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The luminal Ca2+ chelator, TPEN, inhibits NAADP-induced Ca2+ release
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The luminal Ca(2+) chelator, TPEN, inhibits NAADP-induced Ca(2+) release.
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Dual effects of cyclic ADP-ribose on sarcoplasmic reticulum Ca2+ release and storage in cardiac myocytes isolated from guinea-pig and rat ventricle.
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