Diaphragm rescue alone prevents heart dysfunction in dystrophic mice.
Duchenne muscular dystrophy (DMD) is an X-linked recessive disease caused, in most cases, by the complete absence of the 427 kDa cytoskeletal protein, dystrophin. There is no effective treatment, and affected individuals die from respiratory failure and cardiomyopathy by age 30. Here, we investigate...
المؤلفون الرئيسيون: | Crisp, A, Yin, H, Goyenvalle, A, Betts, C, Moulton, H, Seow, Y, Babbs, A, Merritt, T, Saleh, A, Gait, M, Stuckey, D, Clarke, K, Davies, K, Wood, M |
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التنسيق: | Journal article |
اللغة: | English |
منشور في: |
2011
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مواد مشابهة
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Prevention of dystrophic pathology in severely affected dystrophin/utrophin-deficient mice by morpholino-oligomer-mediated exon-skipping.
حسب: Goyenvalle, A, وآخرون
منشور في: (2010) -
Upgrading U7snRNA To Complete Efficient Rescue of Dystrophin by Exon-Skipping in DMD Patients
حسب: Goyenvalle, A, وآخرون
منشور في: (2009) -
Functional rescue of dystrophin-deficient mdx mice by a chimeric peptide-PMO
حسب: Yin, H, وآخرون
منشور في: (2010) -
Rescu of severely affected dystrophin/utrophin deficient mice by morpholino-oligomer mediated exon skipping
حسب: Goyenvalle, A, وآخرون
منشور في: (2010) -
Lipolysis and cyclic AMP response to isoproterenol in diaphragms from control and dystrophic mice.
حسب: N A Abumrad, وآخرون
منشور في: (1980-02-01)